Axonal transport defects are a common phenotype in Drosophila models of ALS

Human molecular genetics

Volumn 25, Issue 12, 2016, Pages 2378-2392

  Baldwin, Katie R ^a   Godena, Vinay K ^a   Hewitt, Victoria L ^a   Whitworth, Alexander J ^a  

^a UNIVERSITY OF CAMBRIDGE   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

C9ORF72 PROTEIN, HUMAN; COPPER ZINC SUPEROXIDE DISMUTASE; DNA BINDING PROTEIN; PROTEIN; RNA BINDING PROTEIN FUS; TDP-43 PROTEIN, HUMAN;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL; AXON; BIOSYNTHESIS; DISEASE MODEL; DROSOPHILA; GENETICS; HUMAN; LARVA; METABOLISM; MITOCHONDRION; MOTONEURON; MUTATION; NERVE FIBER TRANSPORT; PATHOLOGY; TRANSGENIC ANIMAL;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; ANIMALS, GENETICALLY MODIFIED; AXONAL TRANSPORT; AXONS; DISEASE MODELS, ANIMAL; DNA-BINDING PROTEINS; DROSOPHILA; HUMANS; LARVA; MITOCHONDRIA; MOTOR NEURONS; MUTATION; PROTEINS; RNA-BINDING PROTEIN FUS; SUPEROXIDE DISMUTASE-1;

EID: 85016427323     PISSN: None     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddw105     Document Type: Article

References (0)