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Volumn 9, Issue , 2014, Pages 56-
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Interleukin-1β causes excitotoxic neurodegeneration and multiple sclerosis disease progression by activating the apoptotic protein p53
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Author keywords
[No Author keywords available]
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Indexed keywords
INTERLEUKIN 1BETA;
PROTEIN P53;
TUMOR NECROSIS FACTOR;
ANIMAL;
APOPTOSIS;
C57BL MOUSE;
CHEMICALLY INDUCED;
DISEASE COURSE;
DRUG EFFECTS;
EXCITATORY POSTSYNAPTIC POTENTIAL;
FEMALE;
METABOLISM;
MULTIPLE SCLEROSIS;
NERVE DEGENERATION;
PHYSIOLOGY;
SYNAPSE;
ANIMALS;
APOPTOSIS;
DISEASE PROGRESSION;
EXCITATORY POSTSYNAPTIC POTENTIALS;
FEMALE;
INTERLEUKIN-1BETA;
MICE, INBRED C57BL;
MULTIPLE SCLEROSIS;
NERVE DEGENERATION;
SYNAPSES;
TUMOR NECROSIS FACTOR-ALPHA;
TUMOR SUPPRESSOR PROTEIN P53;
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EID: 84990982100
PISSN: None
EISSN: 17501326
Source Type: Journal
DOI: 10.1186/1750-1326-9-56 Document Type: Article |
Times cited : (75)
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References (0)
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