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Volumn 2, Issue 6, 2014, Pages 559-567
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The dose-dependent tumor targeting of antibody-IFNγ fusion proteins reveals an unexpected receptor-trapping mechanism in vivo
a a |
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Author keywords
[No Author keywords available]
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Indexed keywords
ANTINEOPLASTIC AGENT;
CANCER ANTIBODY;
EXTRA DOMAIN A FIBRONECTIN, MOUSE;
F8 MONOCLONAL ANTIBODY;
FIBRONECTIN;
GAMMA INTERFERON;
GAMMA INTERFERON RECEPTOR;
HYBRID PROTEIN;
INTERFERON RECEPTOR;
MONOCLONAL ANTIBODY;
TUMOR MARKER;
ANIMAL;
BIOAVAILABILITY;
C57BL MOUSE;
DOSE RESPONSE;
FEMALE;
HAMSTER;
IMMUNOLOGY;
MALE;
METABOLISM;
NEOPLASMS;
TUMOR CELL CULTURE;
ANIMALS;
ANTIBODIES, MONOCLONAL;
ANTIBODIES, NEOPLASM;
ANTINEOPLASTIC AGENTS;
BIOLOGICAL AVAILABILITY;
BIOMARKERS, TUMOR;
CRICETINAE;
DOSE-RESPONSE RELATIONSHIP, IMMUNOLOGIC;
FEMALE;
FIBRONECTINS;
INTERFERON-GAMMA;
MALE;
MICE, INBRED C57BL;
NEOPLASMS;
RECEPTORS, INTERFERON;
RECOMBINANT FUSION PROTEINS;
TUMOR CELLS, CULTURED;
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EID: 84990001693
PISSN: None
EISSN: 23266074
Source Type: Journal
DOI: 10.1158/2326-6066.CIR-13-0182 Document Type: Article |
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Times cited : (32)
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References (0)
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