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Volumn 6, Issue 5, 2016, Pages

Comparison of intramedullary myeloma and corresponding extramedullary soft tissue plasmacytomas using genetic mutational panel analyses

Author keywords

[No Author keywords available]

Indexed keywords

TRANSCRIPTOME; TUMOR MARKER;

EID: 84983663787     PISSN: None     EISSN: 20445385     Source Type: Journal    
DOI: 10.1038/BCJ.2016.35     Document Type: Letter
Times cited : (25)

References (15)
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  • 2
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  • 3
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  • 4
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    • Molecular pathogenesis of multiple myeloma
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  • 5
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  • 7
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    • Rasmussen T, Kuehl M, Lodahl M, Johnsen HE, Dahl IMS. Possible roles for activating RAS mutations in the MGUS to MM transition and in the intramedullary to extramedullary transition in some plasma cell tumors. Blood 2005; 105: 317–323.
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    • Rasmussen, T.1    Kuehl, M.2    Lodahl, M.3    Johnsen, H.E.4    Dahl, I.M.S.5
  • 11
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    • Mutation of NRAS but not KRAS significantly reduces myeloma sensitivity to single-agent bortezomib therapy
    • Mulligan G, Lichter DI, Di Bacco A, Blakemore SJ, Berger A, Koenig E et al. Mutation of NRAS but not KRAS significantly reduces myeloma sensitivity to single-agent bortezomib therapy. Blood 2014; 123: 632–639.
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    • p53 gene mutations in multiple myeloma are associated with advanced forms of malignancy
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  • 15
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    • Soft-tissue plasmacytomas in multiple myeloma: Incidence, mechanisms of extra-medullary spread, and treatment approach
    • Bladé J, Fernández De Larrea C, Rosiñol L, Cibeira MT, Jimènez R, Powles R. Soft-tissue plasmacytomas in multiple myeloma: Incidence, mechanisms of extra-medullary spread, and treatment approach. J Clin Oncol 2011; 29: 3805–3812.
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.