Tau pathogenesis is promoted by Aβ1-42 but not Aβ1-40

Molecular neurodegeneration

Volumn 9, Issue , 2014, Pages 52-

  Hu, Xiaoyan ^a   Li, Xiaoling ^a   Zhao, Mingrui ^a   Gottesdiener, Andrew ^a   Luo, Wenjie ^a   Paul, Steven ^a  

^a WEILL CORNELL MEDICAL COLLEGE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID BETA-PROTEIN (1-42); MAPT PROTEIN, MOUSE; PEPTIDE FRAGMENT; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL; CONFOCAL MICROSCOPY; DISEASE MODEL; GENETICS; GENOTYPE; METABOLISM; MOUSE; PATHOLOGY; PHOSPHORYLATION; PHYSIOLOGY; PROCEDURES; TRANSGENIC ANIMAL;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; ANIMALS, GENETICALLY MODIFIED; DISEASE MODELS, ANIMAL; GENOTYPE; MICE; MICROSCOPY, CONFOCAL; PEPTIDE FRAGMENTS; PHOSPHORYLATION; TAU PROTEINS;

EID: 84965185095     PISSN: None     EISSN: 17501326     Source Type: Journal    
DOI: 10.1186/1750-1326-9-52     Document Type: Article

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