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Volumn 12, Issue 2, 2016, Pages 424-425
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Cellular processes underlying cerebral cavernous malformations: Autophagy as another point of view
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Author keywords
Autophagy; CCM; Endothelial to mesenchymal transition (EndMt); MTOR; Pharmacological therapy; ROS
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Indexed keywords
ACETYLCYSTEINE;
BETA CATENIN;
BONE MORPHOGENETIC PROTEIN;
COLECALCIFEROL;
HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE INHIBITOR;
MAMMALIAN TARGET OF RAPAMYCIN;
MAMMALIAN TARGET OF RAPAMYCIN INHIBITOR;
MICROTUBULE ASSOCIATED PROTEIN;
NOTCH RECEPTOR;
PROPRANOLOL;
PROTEIN AGGREGATE;
PROTEIN MAP1LC3B;
PROTEIN P62;
RAPAMYCIN;
REACTIVE OXYGEN METABOLITE;
RHO KINASE;
SULINDAC;
TORIN 1;
TRANSFORMING GROWTH FACTOR BETA;
UNCLASSIFIED DRUG;
ARTICLE;
AUTOPHAGY;
BRAIN HEMORRHAGE;
CAVERNOUS HEMANGIOMA;
CCM2 GENE;
CCM3 GENE;
DEGRADATION;
ENDOTHELIUM CELL;
GENE;
HUMAN;
KRIT1 GENE;
PATHOGENESIS;
PDCD10 GENE;
SIGNAL TRANSDUCTION;
TUMOR VASCULARIZATION;
BIOLOGICAL MODEL;
BRAIN HEMANGIOMA;
PATHOLOGY;
AUTOPHAGY;
HEMANGIOMA, CAVERNOUS, CENTRAL NERVOUS SYSTEM;
HUMANS;
MODELS, BIOLOGICAL;
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EID: 84964475949
PISSN: 15548627
EISSN: 15548635
Source Type: Journal
DOI: 10.1080/15548627.2015.1125073 Document Type: Article |
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Times cited : (25)
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References (0)
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