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Volumn 12, Issue 2, 2015, Pages 2323-2328
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Chloroquine exerts neuroprotection following traumatic brain injury via suppression of inflammation and neuronal autophagic death
a a a a a a a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
BIOLOGICAL MARKER;
CELL NUCLEUS ANTIGEN;
CHLOROQUINE;
INTERLEUKIN 1BETA;
LC3 PROTEIN, RAT;
MICROTUBULE ASSOCIATED PROTEIN;
NERVE PROTEIN;
NEUN PROTEIN, RAT;
NEUROPROTECTIVE AGENT;
TUMOR NECROSIS FACTOR;
ANIMAL;
ANTAGONISTS AND INHIBITORS;
AUTOPHAGY;
BIOSYNTHESIS;
BRAIN EDEMA;
BRAIN INJURIES;
DISEASE MODEL;
DRUG ADMINISTRATION;
DRUG EFFECTS;
GENE EXPRESSION;
GENETICS;
HIPPOCAMPUS;
INFLAMMATION;
INJURY SCALE;
INTRAPERITONEAL DRUG ADMINISTRATION;
MAZE TEST;
METABOLISM;
NERVE CELL;
PATHOLOGY;
RAT;
SECRETION (PROCESS);
SPRAGUE DAWLEY RAT;
ANIMALS;
ANTIGENS, NUCLEAR;
AUTOPHAGY;
BIOMARKERS;
BRAIN EDEMA;
BRAIN INJURIES;
CHLOROQUINE;
DISEASE MODELS, ANIMAL;
DRUG ADMINISTRATION SCHEDULE;
GENE EXPRESSION;
HIPPOCAMPUS;
INFLAMMATION;
INJECTIONS, INTRAPERITONEAL;
INTERLEUKIN-1BETA;
MAZE LEARNING;
MICROTUBULE-ASSOCIATED PROTEINS;
NERVE TISSUE PROTEINS;
NEURONS;
NEUROPROTECTIVE AGENTS;
RATS;
RATS, SPRAGUE-DAWLEY;
TRAUMA SEVERITY INDICES;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 84955562920
PISSN: None
EISSN: 17913004
Source Type: Journal
DOI: 10.3892/mmr.2015.3611 Document Type: Article |
Times cited : (46)
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References (0)
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