The authors reply

New England Journal of Medicine

Volumn 373, Issue 18, 2015, Pages 1787-1788

  Gumz, Michelle L ^a   Rabinowitz, Lawrence ^b   Wingo, Charles S ^c  

^a UNIVERSITY OF FLORIDA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALDOSTERONE; ALDOSTERONE SYNTHASE; POTASSIUM; SODIUM; SODIUM CHLORIDE COTRANSPORTER;

ALDOSTERONE BLOOD LEVEL; BLOOD PRESSURE; CIRCADIAN RHYTHM; HUMAN; HYPERKALEMIA; KALIURESIS; KIDNEY COLLECTING TUBULE; KIDNEY DISTAL TUBULE; KIDNEY PERFUSION; LETTER; NEPHRON; NONHUMAN; POSITIVE FEEDBACK; POTASSIUM BLOOD LEVEL; POTASSIUM METABOLISM; PRIORITY JOURNAL; SODIUM ABSORPTION; SODIUM BALANCE; SODIUM RETENTION; HOMEOSTASIS; METABOLISM; PHYSIOLOGY;

CIRCADIAN CLOCKS; HOMEOSTASIS; HUMANS; POTASSIUM;

EID: 84946096653     PISSN: 00284793     EISSN: 15334406     Source Type: Journal    
DOI: 10.56/NEJMc1509656     Document Type: Letter

References (3)

1. Sorensen MV, Grossmann S, Roesinger M, et al. Rapid dephosphorylation of the renal sodium chloride cotransporter in response to oral potassium intake in mice. Kidney Int 2013; 83:811-24.
2. Stanton BA, Giebisch GH. Potassium transport by the renal distal tubule: effects of potassium loading. Am J Physiol 1982; 243:F487-F493.
3. Todkar A, Picard N, Loffing-Cueni D, et al. Mechanisms of renal control of potassium homeostasis in complete aldosterone deficiency. J Am Soc Nephrol 2015;26:425-38.