GABA circuitry, cells and molecular regulation in schizophrenia: Life in the graveyard

Schizophrenia Research

Volumn 167, Issue 1-3, 2015, Pages 108-110

  Harrison, Paul J ^a  

^a UNIVERSITY OF OXFORD   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

GLUTAMIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; 4 AMINOBUTYRIC ACID; 4 AMINOBUTYRIC ACID RECEPTOR;

BIPOLAR DISORDER; CELL FUNCTION; DISEASE ASSOCIATION; DNA METHYLATION; DOPAMINERGIC ACTIVITY; EPIGENETICS; GABAERGIC SYSTEM; GAD1 GENE; GENE; GENETIC ASSOCIATION; GENETIC REGULATION; HUMAN; INTERNEURON; MOLECULAR BIOLOGY; NEUROPATHOLOGY; NONHUMAN; NOTE; OSCILLATION; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; SCHIZOAFFECTIVE PSYCHOSIS; SCHIZOPHRENIA; ANIMAL; BRAIN; METABOLISM; PATHOLOGY;

ANIMALS; BRAIN; GABAERGIC NEURONS; GAMMA-AMINOBUTYRIC ACID; HUMANS; SCHIZOPHRENIA;

EID: 84942195521     PISSN: 09209964     EISSN: 15732509     Source Type: Journal    
DOI: 10.1016/j.schres.2015.02.003     Document Type: Note

References (2)

1. Belforte J.E., Zsiros V., Sklar E.R., Jiang Z., Yu G., Li Y., Quinlan E.M., Nakazawa K. Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes. Nat. Neurosci. 2010, 13(1):76-83.
2. Benes F.M. Building models for postmortem abnormalities in hippocampus of schizophrenics. Schizophr. Res. 2015, 167(1--3):73-83. (in this issue).