A reninssance in primary aldosteronism testing: Obstacles and opportunities for screening, diagnosis, and management

Clinical Chemistry

Volumn 61, Issue 8, 2015, Pages 1022-1027

  Raizman, Joshua E ^a   Diamandis, Eleftherios P ^a,b,c   Holmes, Daniel ^d,e   Stowasser, Michael ^f   Auchus, Richard ^g   Cavalier, Etienne ^h  

^a UNIVERSITY OF TORONTO   (Canada)

^b UNIVERSITY HEALTH NETWORK   (Canada)

^c ST PAUL S HOSPITAL   (Canada)

^d UNIVERSITY OF BRITISH COLUMBIA   (Canada)

^e UniversityofQueenslandSchoolofMedicine   (Australia)

^f UNIVERSITY OF MICHIGAN MEDICAL SCHOOL   (United States)

^g UNIVERSITY OF LIÈGE   (Belgium)

^h MOUNT SINAI HOSPITAL   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ALDOSTERONE; ANGIOTENSIN II; PRORENIN; RENIN; ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM); ATP1A1 PROTEIN, HUMAN; ATP2B3 PROTEIN, HUMAN; CACNA1D PROTEIN, HUMAN; CALCIUM CHANNEL L TYPE; FLUDROCORTISONE; G PROTEIN COUPLED INWARDLY RECTIFYING POTASSIUM CHANNEL; KCNJ5 PROTEIN, HUMAN; PLASMA MEMBRANE CALCIUM TRANSPORTING ADENOSINE TRIPHOSPHATASE;

ALDOSTERONE BLOOD LEVEL; ALDOSTERONE RELEASE; ALDOSTERONE URINE LEVEL; CROSS REACTION; HORMONE SYNTHESIS; HUMAN; HYPERTENSION; IMMUNOASSAY; MOLECULAR DIAGNOSIS; NOTE; PRIMARY HYPERALDOSTERONISM; SALT LOADING; SCREENING; TANDEM MASS SPECTROMETRY; BLOOD; COMPLICATION; DIAGNOSTIC USE; GENETICS; HYPERALDOSTERONISM; MUTATION; PROCEDURES; REFERENCE VALUE; SENSITIVITY AND SPECIFICITY; STANDARDS;

ALDOSTERONE; CALCIUM CHANNELS, L-TYPE; FLUDROCORTISONE; G PROTEIN-COUPLED INWARDLY-RECTIFYING POTASSIUM CHANNELS; HUMANS; HYPERALDOSTERONISM; HYPERTENSION; IMMUNOASSAY; MUTATION; PLASMA MEMBRANE CALCIUM-TRANSPORTING ATPASES; REFERENCE VALUES; RENIN; SENSITIVITY AND SPECIFICITY; SODIUM-POTASSIUM-EXCHANGING ATPASE;

EID: 84938819367     PISSN: 00099147     EISSN: 15308561     Source Type: Journal    
DOI: 10.1373/clinchem.2015.242990     Document Type: Note

References (2)

1. Professor of Internal Medicine and Director, Fellowship Program, Division of Metabolism, Endocrinology & Diabetes, Department of Internal Medicine University of Michigan Medical School, Ann Arbor, MI;.
2. Human genes: KCNJ5, potassium inwardly-rectifying channel subfamily J member 5;.