Diallyl trisulfide protects against high glucose-induced cardiac apoptosis by stimulating the production of cystathionine gamma-lyase-derived hydrogen sulfide

International Journal of Cardiology

Volumn 195, Issue , 2015, Pages 300-310

  Tsai, Cheng Yen ^a,b   Wen, Su Ying ^c,d   Shibu, Marthandam Asokan ^b   Yang, Yao Chih ^b   Peng, Hanjing ^e   Wang, Binghe ^e   Wei, Yu Min ^b   Chang, Hung Yu ^f   Lee, Cheng Yu ^c   Huang, Chih Yang ^b,g,h   Kuo, Wei Wen ^b  

^a CHINA MEDICAL UNIVERSITY BEIGANG HOSPITAL   (Taiwan)

^b CHINA MEDICAL UNIVERSITY   (Taiwan)

^c TAIPEI CITY HOSPITAL   (Taiwan)

^d NURSING AND MANAGEMENT COLLEGE   (Taiwan)

^e GEORGIA STATE UNIVERSITY   (United States)

^f CHANG GUNG MEMORIAL HOSPITAL   (Taiwan)

^g CHINA MEDICAL UNIVERSITY HOSPITAL   (Taiwan)

^h ASIA UNIVERSITY   (Taiwan)

Author keywords

Apoptosis; Cystathionine gamma lyase (CSE); Diallyl trisulfide (DATS); High glucose (HG); Hydrogen sulfide (H2S); Reactive oxygen species (ROS)

Indexed keywords

CASPASE 3; CYSTATHIONINE GAMMA LYASE; DIALLYL TRISULFIDE; GLUCOSE; HYDROGEN SULFIDE; PROTEIN KINASE B; REACTIVE OXYGEN METABOLITE; SOMATOMEDIN C RECEPTOR; ALLYL COMPOUND; CARDIOTONIC AGENT; SULFIDE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; DIABETES MELLITUS; ENZYME ACTIVATION; ENZYME MECHANISM; ENZYME SYNTHESIS; HEART INJURY; HEART MUSCLE CELL; HEART PROTECTION; HYPERGLYCEMIA; MALE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; SIGNAL TRANSDUCTION; ANIMAL; BIOLOGICAL MODEL; CARDIOMYOPATHIES; CELL LINE; CELL PROTECTION; DISEASE MODEL; DRUG EFFECTS; GARLIC; HUMAN; METABOLISM; WISTAR RAT;

ALLYL COMPOUNDS; ANIMALS; APOPTOSIS; CARDIOMYOPATHIES; CARDIOTONIC AGENTS; CELL LINE; CYSTATHIONINE GAMMA-LYASE; CYTOPROTECTION; DIABETES COMPLICATIONS; DISEASE MODELS, ANIMAL; GARLIC; GLUCOSE; HUMANS; HYDROGEN SULFIDE; MALE; MODELS, CARDIOVASCULAR; MYOCYTES, CARDIAC; RATS; RATS, WISTAR; SIGNAL TRANSDUCTION; SULFIDES;

EID: 84937611169     PISSN: 01675273     EISSN: 18741754     Source Type: Journal    
DOI: 10.1016/j.ijcard.2015.05.111     Document Type: Article

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