The authors reply

New England Journal of Medicine

Volumn 373, Issue 2, 2015, Pages 195-

  Beaugerie, Laurent ^a   Itzkowitz, Steven H ^b  

^a SORBONNE UNIVERSITÉ   (France)

^b MOUNT SINAI SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE;

ADAPTIVE IMMUNITY; CARCINOGENESIS; COLITIS; COLORECTAL CANCER; DISEASE ASSOCIATION; ENTERITIS; EPITHELIUM CELL; HUMAN; IMMUNOPATHOGENESIS; INFLAMMATORY BOWEL DISEASE; INNATE IMMUNITY; LETTER; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; TH17 CELL; COLORECTAL NEOPLASMS; COMPLICATION;

COLORECTAL NEOPLASMS; HUMANS; INFLAMMATORY BOWEL DISEASES;

EID: 84936751961     PISSN: 00284793     EISSN: 15334406     Source Type: Journal    
DOI: 10.1056/NEJMc1505689     Document Type: Letter

References (5)

1. Ullman TA, Itzkowitz SH. Intestinal inflammation and cancer. Gastroenterology 2011;140:1807-16.
2. Troncone E, Marafini I, Pallone F, Monteleone G. Th17 cytokines in inflammatory bowel diseases: discerning the good from the bad. Int Rev Immunol 2013;32:526-33.
3. De Simone V, Franz? E, Ronchetti G, et al. Th17-type cytokines, IL-6 and TNF-α synergistically activate STAT3 and NF-kB to promote colorectal cancer cell growth. Oncogene 2014 September 1 (Epub ahead of print).
4. Martin M, Kesselring RK, Saidou B, et al. RORγt(+) hematopoietic cells are necessary for tumor cell proliferation during colitis-associated tumorigenesis in mice. Eur J Immunol 2015; 45:1667-79.
5. Punkenburg E, Vogler T, Büttner M, et al. Batf-dependent Th17 cells critically regulate IL-23 driven colitis-associated colon cancer. Gut 2015 April 2 (Epub ahead of print).