Reply to Wilkinson: Minor role of programmed methylation and demethylation in mammalian development

Proceedings of the National Academy of Sciences of the United States of America

Volumn 112, Issue 17, 2015, Pages E2117-

  Bestor, Timothy H ^a   Edwards, John R ^b   Boulard, Mathieu ^a  

^a New York Presbyterian Columbia University Medical Center   (United States)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MAMMALIA;

ANIMAL; DNA METHYLATION; GENE EXPRESSION REGULATION; GENETIC EPIGENESIS; GROWTH, DEVELOPMENT AND AGING; MAMMAL; PHYSIOLOGY;

ANIMALS; DNA METHYLATION; EPIGENESIS, GENETIC; GENE EXPRESSION REGULATION, DEVELOPMENTAL; MAMMALS;

EID: 84928528588     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1504199112     Document Type: Letter

References (6)

1. Wilkinson MF (2015) Evidence that DNA methylation engenders dynamic gene regulation. Proc Natl Acad Sci USA 112:E2116.
2. Bestor TH, Edwards JR, Boulard M (2014) Notes on the role of dynamic DNA methylation in mammalian development. Proc Natl Acad Sci USA, 10.1073/pnas.1415301111.
3. Kobayashi H, et al. (2012) Contribution of intragenic DNA methylation in mouse gametic DNA methylomes to establish oocyte-specific heritable marks. PLoS Genet 8(1):e1002440.
4. Bourc'his D, Xu GL, Lin CS, Bollman B, Bestor TH (2001) Dnmt3L and the establishment of maternal genomic imprints. Science 294(5551):2536-2539.
5. Pevny L, et al. (1991) Erythroid differentiation in chimaeric mice blocked by a targeted mutation in the gene for transcription factor GATA-1. Nature 349(6306):257-260.
6. Dawlaty MM, et al. (2011) Tet1 is dispensable for maintaining pluripotency and its loss is compatible with embryonic and postnatal development. Cell Stem Cell 9(2):166-175.