ALLELE;
ARTICLE;
CANCER CHEMOTHERAPY;
CANCER PATIENT;
CANCER SURVIVAL;
COLORECTAL CANCER;
GENE MUTATION;
HUMAN;
IN VITRO STUDY;
JAPANESE (PEOPLE);
LIMIT OF DETECTION;
ONCOGENE K RAS;
ONCOGENE N RAS;
ONCOLOGY;
OVERALL SURVIVAL;
PRACTICE GUIDELINE;
PRIORITY JOURNAL;
PROGRESSION FREE SURVIVAL;
THERAPY EFFECT;
ANTAGONISTS AND INHIBITORS;
COLORECTAL NEOPLASMS;
DNA SEQUENCE;
GENETIC SCREENING;
GENETICS;
IMMUNOLOGY;
JAPAN;
MUTATION;
PROCEDURES;
STAINING;
Japanese Society of Medical Oncology Clinical Guidelines: RAS (KRAS/NRAS) mutation testing in colorectal cancer patients
(1 screen). [Cited 10 Apr .] Available from URL:.
Japanese Society of Medical Oncology. Japanese Society of Medical Oncology Clinical Guidelines: RAS (KRAS/NRAS) mutation testing in colorectal cancer patients (1 screen). [Cited 10 Apr 2014.] Available from URL: http://www.jsmo.or.jp/file/dl/newsj/1288.pdf.
(2014)
2
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Panitumumab-FOLFOX4 treatment and RAS mutations in colorectal cancer
Douillard JY, Oliner KS, Siena S et al. Panitumumab-FOLFOX4 treatment and RAS mutations in colorectal cancer. N Engl J Med 2013; 369: 1023-34.
PEAK: a randomized, multicenter phase II study of panitumumab plus modified fluorouracil, leucovorin, and oxaliplatin (mFOLFOX6) or bevacizumab plus mFOLFOX6 in patients with previously untreated, unresectable, wild-type KRAS exon 2 metastatic colorectal cancer
Schwartzberg LS, Rivera F, Karthaus M et al. PEAK: a randomized, multicenter phase II study of panitumumab plus modified fluorouracil, leucovorin, and oxaliplatin (mFOLFOX6) or bevacizumab plus mFOLFOX6 in patients with previously untreated, unresectable, wild-type KRAS exon 2 metastatic colorectal cancer. J Clin Oncol 2014; 32: 2240-7.
K-ras mutations and benefit from cetuximab in advanced colorectal cancer
Karapetis CS, Khambata-Ford S, Jonker DJ et al. K-ras mutations and benefit from cetuximab in advanced colorectal cancer. N Engl J Med 2008; 359: 17-65.
Association of KRAS G13D tumor mutations with outcome in patients with metastatic colorectal cancer treated with first-line chemotherapy with or without cetuximab
Tejpar S, Celik I, Schlichting M et al. Association of KRAS G13D tumor mutations with outcome in patients with metastatic colorectal cancer treated with first-line chemotherapy with or without cetuximab. J Clin Oncol 2012; 30: 3570-7.
Mutant KRAS codon 12 and 13 alleles in patients with metastatic colorectal cancer: assessment as prognostic and predictive biomarkers of response to panitumumab
Peeters M, Douillard JY, Van Cutsem E et al. Mutant KRAS codon 12 and 13 alleles in patients with metastatic colorectal cancer: assessment as prognostic and predictive biomarkers of response to panitumumab. J Clin Oncol 2013; 31: 759-65.
Effects of KRAS, BRAF, NRAS, and PIK3CA mutations on the efficacy of cetuximab plus chemotherapy in chemotherapy-refractory metastatic colorectal cancer: a retrospective consortium analysis
De Roock W, Claes B, Bernasconi D et al. Effects of KRAS, BRAF, NRAS, and PIK3CA mutations on the efficacy of cetuximab plus chemotherapy in chemotherapy-refractory metastatic colorectal cancer: a retrospective consortium analysis. Lancet Oncol 2010; 11: 753-62.
Clinical validation of a novel multiplex kit for all RAS mutations in colorectal cancer: results of RASKET (RAS KEy Testing) prospective multicenter study
P-0195, WCGIC
Kudo T, Satoh T, Muro K et al. Clinical validation of a novel multiplex kit for all RAS mutations in colorectal cancer: results of RASKET (RAS KEy Testing) prospective multicenter study. WCGIC 2014, P-0195
Concordance of predictive markers for EGFR inhibitors in primary tumors and metastases in colorectal cancer: a review
Baas JM, Krens LL, Guchelaar HJ et al. Concordance of predictive markers for EGFR inhibitors in primary tumors and metastases in colorectal cancer: a review. Oncologist 2011; 16: 1239-49.
KRAS mutation: comparison of testing methods and tissue sampling techniques in colon cancer
Franklin WA, Haney J, Sugita M et al. KRAS mutation: comparison of testing methods and tissue sampling techniques in colon cancer. J Mol Diagn 2010; 12: 43-50.