Mutations in the cytoplasmic domain of P0 reveal a role for PKC-mediated phosphorylation in adhesion and myelination

Journal of Cell Biology

Volumn 155, Issue 4, 2001, Pages 439-445

  Xu, Wenbo ^a   Shy, Michael ^a   Kamholz, John ^a   Elferink, Lisa ^b   Xu, Gang ^c   Lilien, Jack ^c   Balsamo, Janne ^c  

^a WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b Biomedical Marine Institute   (United States)

^c UNIVERSITY OF IOWA   (United States)

Author keywords

Adhesion; Myelination; P0; PKC; RACK1

Indexed keywords

AMINO ACID; BINDING PROTEIN; CELL ADHESION MOLECULE; MUTANT PROTEIN; MYELIN PROTEIN; PROTEIN KINASE C; PROTEIN KINASE C ALPHA; PROTEIN P0; SERINE; UNCLASSIFIED DRUG; ISOENZYME; PEPTIDE; PEPTIDE I; PRKCA PROTEIN, HUMAN; PRKCA PROTEIN, MOUSE;

AMINO ACID SEQUENCE; ANIMAL CELL; ARTICLE; CELL ADHESION; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME INHIBITION; GENE DELETION; GENE MUTATION; HEREDITARY MOTOR SENSORY NEUROPATHY; HUMAN; HUMAN CELL; IMMUNOPRECIPITATION; MOUSE; MYELINATION; NONHUMAN; POINT MUTATION; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN FUNCTION; PROTEIN MOTIF; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; ANIMAL; BINDING SITE; CYTOPLASM; DEMYELINATING DISEASE; GENETICS; HELA CELL LINE; L CELL LINE; METABOLISM; MOLECULAR GENETICS; PHOSPHORYLATION; PHYSIOLOGY;

ANIMALIA;

AMINO ACID SEQUENCE; AMINO ACIDS; ANIMALS; BINDING SITES; CELL ADHESION; CHARCOT-MARIE-TOOTH DISEASE; CYTOPLASM; DEMYELINATING DISEASES; HELA CELLS; HUMANS; ISOENZYMES; L CELLS (CELL LINE); MICE; MOLECULAR SEQUENCE DATA; MYELIN P0 PROTEIN; PEPTIDES; PHOSPHORYLATION; PROTEIN KINASE C; PROTEIN KINASE C-ALPHA; SEQUENCE DELETION;

EID: 84924094149     PISSN: 00219525     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (30)

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