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Volumn 103, Issue 2, 2015, Pages 528-534.e13

Follow-up to genome-wide linkage and admixture mapping studies implicates components of the extracellular matrix in susceptibility to and size of uterine fibroids

Author keywords

collagen; extracellular matrix; fibroids; NIEHS cohort; Polymorphism; susceptibility

Indexed keywords

ADULT; AFRICAN AMERICAN; ARTICLE; CANCER RISK; CANCER SUSCEPTIBILITY; CASE CONTROL STUDY; COL13A GENE; COL6A3 GENE; CONTROLLED STUDY; CROSS-SECTIONAL STUDY; ETHNIC DIFFERENCE; EUROPEAN AMERICAN; EXTRACELLULAR MATRIX; FEMALE; FOLLOW UP; GENE EXPRESSION REGULATION; GENE LINKAGE DISEQUILIBRIUM; GENE MAPPING; GENE REPLICATION; GENETIC ASSOCIATION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; HIGH RISK POPULATION; HUMAN; LINKAGE ANALYSIS; MAJOR CLINICAL STUDY; MANNOSIDASE ALPHA CLASS 1C GENE; MISSENSE MUTATION; NORTH AMERICAN; PREMENOPAUSE; PRIORITY JOURNAL; RHO GTPASE ACTIVATING PROTEIN 26 GENE; SINGLE NUCLEOTIDE POLYMORPHISM; TUMOR GENE; TUMOR VOLUME; UTERUS MYOMA; CHROMOSOMAL MAPPING; GENETIC LINKAGE; GENETIC PREDISPOSITION; GENETICS; GENOME-WIDE ASSOCIATION STUDY; LEIOMYOMA; MIDDLE AGED; PATHOLOGY; PHYSIOLOGY; PROCEDURES; RANDOMIZED CONTROLLED TRIAL; UTERINE NEOPLASMS;

EID: 84923197061     PISSN: 00150282     EISSN: 15565653     Source Type: Journal    
DOI: 10.1016/j.fertnstert.2014.10.025     Document Type: Article
Times cited : (20)

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