Diabetes: Epigenetic changes lead to impaired wound healing in patients with T2DM

Nature Reviews Endocrinology

Volumn 11, Issue 2, 2015, Pages 65-

  Sidaway, Peter ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

HISTONE H3; INTERLEUKIN 12;

CHRONIC INFLAMMATION; CHRONIC WOUND; DIABETIC PATIENT; EPIGENETICS; GENE EXPRESSION; GLUCOSE INTOLERANCE; HEMATOPOIETIC STEM CELL; HISTONE METHYLATION; HUMAN; MACROPHAGE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; NOTE; PHENOTYPE; PRIORITY JOURNAL; PROMOTER REGION; WOUND HEALING; ANIMAL; BONE MARROW CELL; GENETIC EPIGENESIS; METABOLISM; PHYSIOLOGY; STEM CELL;

ANIMALS; BONE MARROW CELLS; DIABETES MELLITUS, TYPE 2; EPIGENESIS, GENETIC; HUMANS; STEM CELLS; WOUND HEALING;

EID: 84923091994     PISSN: 17595029     EISSN: 17595037     Source Type: Journal    
DOI: 10.1038/nrendo.2014.207     Document Type: Note

References (1)

1. Gallagher, K. A. et al. Epigenetic changes in bone marrow progenitor cells influence the inflammatory phenotype and alter wound healing in type 2 diabetes. Diabetes doi:10.2337/db14-0872