Reply: The BIM deletion polymorphism cannot account for intrinsic TKI resistance of Chinese individuals with chronic myeloid leukemia

Nature Medicine

Volumn 20, Issue 10, 2014, Pages 1090-1091

  Ong, S Tiong ^a,b,c,d   Chuah, Charles T H ^a,b   Ko, Tun Kiat ^a   Hillmer, Axel M ^e   Lim, Wan Teck ^c  

^a DUKE NUS MEDICAL SCHOOL   (Singapore)

^b SINGAPORE GENERAL HOSPITAL   (Singapore)

^c DIVISION OF MEDICAL ONCOLOGY   (Singapore)

^d DUKE UNIVERSITY MEDICAL CENTER   (United States)

^e GENOME INSTITUTE OF SINGAPORE   (Singapore)

Author keywords

[No Author keywords available]

Indexed keywords

BIM PROTEIN; IMATINIB; APOPTOSIS REGULATORY PROTEIN; MEMBRANE PROTEIN; ONCOPROTEIN; PROTEIN KINASE INHIBITOR;

CHINESE; CHRONIC MYELOID LEUKEMIA; CLINICAL FEATURE; GENE DELETION; HETEROZYGOTE; HUMAN; LETTER; MAJOR CLINICAL STUDY; PRIORITY JOURNAL; PROTEIN POLYMORPHISM; TREATMENT RESPONSE; WILD TYPE; APOPTOSIS; DRUG EFFECTS; DRUG RESISTANCE; FEMALE; GENETIC POLYMORPHISM; GENETICS; LUNG TUMOR; MALE; NON SMALL CELL LUNG CANCER;

APOPTOSIS; APOPTOSIS REGULATORY PROTEINS; CARCINOMA, NON-SMALL-CELL LUNG; DRUG RESISTANCE, NEOPLASM; FEMALE; HUMANS; LEUKEMIA, MYELOGENOUS, CHRONIC, BCR-ABL POSITIVE; LUNG NEOPLASMS; MALE; MEMBRANE PROTEINS; POLYMORPHISM, GENETIC; PROTEIN KINASE INHIBITORS; PROTO-ONCOGENE PROTEINS; SEQUENCE DELETION;

EID: 84921960257     PISSN: 10788956     EISSN: 1546170X     Source Type: Journal    
DOI: 10.1038/nm.3652     Document Type: Letter

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