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Volumn 25, Issue 5, 2014, Pages 917-918

Deciphering root causes of intrinsic BRAF inhibitor resistance in melanoma: ushering in a new genomics case reports feature for Annals of Oncology

Author keywords

[No Author keywords available]

Indexed keywords

ANTINEOPLASTIC AGENT; B RAF KINASE; BRAF PROTEIN, HUMAN;

EID: 84919445438     PISSN: 09237534     EISSN: 15698041     Source Type: Journal    
DOI: 10.1093/annonc/mdu060     Document Type: Editorial
Times cited : (5)

References (11)
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    • Improved survival with vemurafenib in melanoma with BRAF V600E mutation
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    • Which drug, and when, for patients with BRAF-mutant melanoma?
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    • The genetic landscape of clinical resistance to RAF inhibition in metastatic melanoma
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  • 7
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    • Acquired resistance and clonal evolution in melanoma during BRAF inhibitor therapy
    • Shi, H., Hugo, W., Kong, X., et al. Acquired resistance and clonal evolution in melanoma during BRAF inhibitor therapy. Cancer Discov 4 (2014), 80–93.
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    • Shi, H.1    Hugo, W.2    Kong, X.3
  • 8
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    • Identification of multiple mechanisms of resistance to vemurafenib in a patient with BRAFV600E-mutated cutaneous melanoma successfully rechallenged after progression
    • Romano, E., Pradervand, S., Paillusson, A., et al. Identification of multiple mechanisms of resistance to vemurafenib in a patient with BRAFV600E-mutated cutaneous melanoma successfully rechallenged after progression. Clin Cancer Res 19 (2013), 5749–5757.
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.