A new kind of cell suicide: Mechanisms and functions of programmed necrosis

Trends in Biochemical Sciences

Volumn 39, Issue 12, 2014, Pages 587-593

  Sun, Liming ^a   Wang, Xiaodong ^b  

^a INSTITUTE OF BIOCHEMISTRY AND CELL BIOLOGY   (China)

^b NATIONAL INSTITUTE OF BIOLOGICAL SCIENCES   (China)

Author keywords

Mixed lineage kinase domain like protein (MLKL); Necroptosis; Necrosis biomarker; Necrosis inhibitors; Programmed necrosis; Pseudokinase

Indexed keywords

INHIBITOR OF APOPTOSIS PROTEIN; INTERFERON REGULATORY FACTOR 3; MITOCHONDRIAL DNA; MIXED LINEAGE KINASE DOMAIN LIKE PROTEIN; RECEPTOR INTERACTING KINASE 1; RECEPTOR INTERACTING PROTEIN SERINE THREONINE KINASE; UNCLASSIFIED DRUG;

APOPTOSIS; BINDING AFFINITY; CELL DEATH; CELL FUNCTION; CELL MEMBRANE; HUMAN; IMMUNE RESPONSE; IMMUNOREACTIVITY; MEMBRANE DAMAGE; MOLECULAR DYNAMICS; MOLECULAR PATHOLOGY; NECROPTOSIS; NONHUMAN; OLIGOMERIZATION; PROTEIN ANALYSIS; PROTEIN DEGRADATION; PROTEIN DOMAIN; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; PROTEIN STABILITY; REVIEW; SIGNAL TRANSDUCTION; UBIQUITINATION; ANIMAL; BIOLOGICAL MODEL; NECROSIS; NOMENCLATURE;

ANIMALIA;

ANIMALS; APOPTOSIS; CELL DEATH; HUMANS; MODELS, BIOLOGICAL; NECROSIS; SIGNAL TRANSDUCTION; TERMINOLOGY AS TOPIC;

EID: 84918767312     PISSN: 09680004     EISSN: 13624326     Source Type: Journal    
DOI: 10.1016/j.tibs.2014.10.003     Document Type: Review

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