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Volumn 41, Issue 3, 2014, Pages 729-737
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Pharmacological modulation of GSAP reduces amyloid-β levels and tau phosphorylation in a mouse model of Alzheimer's disease with plaques and tangles
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Author keywords
Amyloid ; imatinib; tau protein; transgenic mice; secretase
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Indexed keywords
AMYLOID BETA PROTEIN;
AMYLOID BETA PROTEIN[1-40];
AMYLOID BETA-PROTEIN (1-42);
AMYLOID PRECURSOR PROTEIN;
BENZAMIDE DERIVATIVE;
ENZYME INHIBITOR;
GAMMA-SECRETASE ACTIVATING PROTEIN, MOUSE;
IMATINIB;
PEPTIDE FRAGMENT;
PIPERAZINE DERIVATIVE;
PRESENILIN 1;
PROTEIN;
PSEN1 PROTEIN, HUMAN;
PYRIMIDINE DERIVATIVE;
SARCOSINE;
SARKOSYL;
TAU PROTEIN;
ALZHEIMER DISEASE;
ANALOGS AND DERIVATIVES;
ANIMAL;
COMPLICATION;
DISEASE MODEL;
DRUG EFFECTS;
GENETICS;
HUMAN;
METABOLISM;
MOUSE;
MUTATION;
NEUROBLASTOMA;
NONPARAMETRIC TEST;
PATHOLOGY;
PHOSPHORYLATION;
PLAQUE, AMYLOID;
TRANSGENIC MOUSE;
TUMOR CELL LINE;
ALZHEIMER DISEASE;
AMYLOID BETA-PEPTIDES;
AMYLOID BETA-PROTEIN PRECURSOR;
ANIMALS;
BENZAMIDES;
CELL LINE, TUMOR;
DISEASE MODELS, ANIMAL;
ENZYME INHIBITORS;
HUMANS;
MICE;
MICE, TRANSGENIC;
MUTATION;
NEUROBLASTOMA;
PEPTIDE FRAGMENTS;
PHOSPHORYLATION;
PIPERAZINES;
PLAQUE, AMYLOID;
PRESENILIN-1;
PROTEINS;
PYRIMIDINES;
SARCOSINE;
STATISTICS, NONPARAMETRIC;
TAU PROTEINS;
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EID: 84917739207
PISSN: None
EISSN: 18758908
Source Type: Journal
DOI: 10.3233/JAD-140105 Document Type: Article |
Times cited : (30)
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References (0)
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