Pro-inflammatory interleukin genotypes potentiate early and advanced atherosclerosis differently

Journal of the American College of Cardiology

Volumn 64, Issue 8, 2014, Pages 848-849

  Oikonomou, Evangelos ^b   Siasos, Gerasimos ^b   Tousoulis, Dimitris ^a  

^a HIPPOKRATION HOSPITAL   (Greece)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

BIOLOGICAL MARKER; MICRORNA 29A; SMAD3 PROTEIN;

DOWN REGULATION; HEART INFARCTION; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HUMAN; LETTER; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

CORONARY ARTERY DISEASE; DNA; FEMALE; HUMANS; INTERLEUKIN-1; LIPOPROTEIN(A); MALE; PHOSPHOLIPIDS; POLYMORPHISM, SINGLE NUCLEOTIDE;

EID: 84907960288     PISSN: 07351097     EISSN: 15583597     Source Type: Journal    
DOI: 10.1016/j.jacc.2014.05.043     Document Type: Letter

References (5)

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2. S. Yusuf, S. Hawken, and S. Ounpuu et al. Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study Lancet 364 2004 937 952
3. G. Hatzis, D. Tousoulis, and N. Papageorgiou et al. Combined effects of smoking and interleukin-6 and C-reactive protein genetic variants on endothelial function, inflammation, thrombosis and incidence of coronary artery disease Int J Cardiol 2014 [E-pub ahead of print]
4. A. Qamar, and D.J. Rader Effect of interleukin 1beta inhibition in cardiovascular disease Curr Opin Lipidol 23 2012 548 553
5. P. Zamani, G.G. Schwartz, and A.G. Olsson et al. Inflammatory biomarkers, death, and recurrent nonfatal coronary events after an acute coronary syndrome in the MIRACL study J Am Heart Assoc 2 2013 e003103