|
Volumn 5, Issue , 2014, Pages
|
The CT20 peptide causes detachment and death of metastatic breast cancer cells by promoting mitochondrial aggregation and cytoskeletal disruption.
a a a a a a a a a a a a a |
Author keywords
[No Author keywords available]
|
Indexed keywords
ANTINEOPLASTIC AGENT;
DRUG CARRIER;
FOLIC ACID;
NANOPARTICLE;
PEPTIDE FRAGMENT;
PROTEIN BAX;
ACTIN FILAMENT;
ANIMAL;
ARTICLE;
BREAST TUMOR;
CELL ADHESION;
CELL DEATH;
CELL SURVIVAL;
DRUG EFFECT;
DRUG SCREENING;
FEMALE;
HUMAN;
METABOLISM;
MITOCHONDRIAL DYNAMICS;
MITOCHONDRIAL MEMBRANE POTENTIAL;
MITOCHONDRION;
MOUSE;
NUDE MOUSE;
PATHOLOGY;
TIME;
TUMOR CELL LINE;
TUMOR VOLUME;
ACTIN CYTOSKELETON;
ANIMALS;
ANTINEOPLASTIC AGENTS;
BCL-2-ASSOCIATED X PROTEIN;
BREAST NEOPLASMS;
CELL ADHESION;
CELL DEATH;
CELL LINE, TUMOR;
CELL SURVIVAL;
DRUG CARRIERS;
FEMALE;
FOLIC ACID;
HUMANS;
MEMBRANE POTENTIAL, MITOCHONDRIAL;
MICE;
MICE, NUDE;
MITOCHONDRIA;
MITOCHONDRIAL DYNAMICS;
NANOPARTICLES;
PEPTIDE FRAGMENTS;
TIME FACTORS;
TUMOR BURDEN;
XENOGRAFT MODEL ANTITUMOR ASSAYS;
|
EID: 84907958757
PISSN: None
EISSN: 20414889
Source Type: Journal
DOI: 10.1038/cddis.2014.225 Document Type: Article |
Times cited : (31)
|
References (0)
|