-
1
-
-
77955050878
-
Arsenic exposure from drinking water, and all-cause and chronic-disease mortalities in Bangladesh (HEALS): A prospective cohort study
-
Argos M, Kalra T, Rathouz PJ, Chen Y, Pierce B, et al. (2010). Arsenic exposure from drinking water, and all-cause and chronic-disease mortalities in Bangladesh (HEALS): a prospective cohort study. Lancet 376: 252-258.
-
(2010)
Lancet
, vol.376
, pp. 252-258
-
-
Argos, M.1
Kalra, T.2
Rathouz, P.J.3
Chen, Y.4
Pierce, B.5
-
2
-
-
77952956992
-
Low-dose synergistic immunosuppression of T-dependent antibody responses by polycyclic aromatic hydrocarbons and arsenic in C57BL/6J murine spleen cells
-
2010 Jun 15
-
Li Q, Lauer FT, Liu KJ, Hudson LG, Burchiel SW (2010). Low-dose synergistic immunosuppression of T-dependent antibody responses by polycyclic aromatic hydrocarbons and arsenic in C57BL/6J murine spleen cells. Toxicol Appl Pharmacol. 2010 Jun 15; 245(3): 344-351.
-
(2010)
Toxicol Appl Pharmacol
, vol.245
, Issue.3
, pp. 344-351
-
-
Li, Q.1
Lauer, F.T.2
Liu, K.J.3
Hudson, L.G.4
Burchiel, S.W.5
-
3
-
-
84899531284
-
Arsenite selectively inhibits mouse bone marrow lymphoid progenitor cell development in vivo and in vitro and suppresses humoral immunity in vivo
-
Apr 8
-
Ezeh PC, Lauer FT, MacKenzie D, McClain S, Liu KJ, et al. (2014). Arsenite selectively inhibits mouse bone marrow lymphoid progenitor cell development in vivo and in vitro and suppresses humoral immunity in vivo. PLoS One. Apr 8; 9(4): e93920. doi: 10.1371/journal.pone.0093920.
-
(2014)
PLoS One
, vol.9
, Issue.4
, pp. e93920
-
-
Ezeh, P.C.1
Lauer, F.T.2
MacKenzie, D.3
McClain, S.4
Liu, K.J.5
-
4
-
-
33646265271
-
Assessment of lymphocyte subpopulations and cytokine secretion in children exposed to arsenic
-
Soto-Peña GA, Luna AL, Acosta-Saavedra L, Conde P, López-Carrillo L, et al. (2006). Assessment of lymphocyte subpopulations and cytokine secretion in children exposed to arsenic. FASEB J. 20: 779-781.
-
(2006)
FASEB J.
, vol.20
, pp. 779-781
-
-
Soto-Peña, G.A.1
Luna, A.L.2
Acosta-Saavedra, L.3
Conde, P.4
López-Carrillo, L.5
-
5
-
-
52649095364
-
Analysis of T-cell proliferation and cytokine secretion in the individuals exposed to arsenic
-
Biswas R, Ghosh P, Banerjee N, Das JK, Sau T, et al. (2008). Analysis of T-cell proliferation and cytokine secretion in the individuals exposed to arsenic. Hum Exp Toxicol. 27: 381-386.
-
(2008)
Hum Exp Toxicol.
, vol.27
, pp. 381-386
-
-
Biswas, R.1
Ghosh, P.2
Banerjee, N.3
Das, J.K.4
Sau, T.5
-
6
-
-
85067776065
-
Inorganic arsenic effects on human lymphocyte stimulation and proliferation
-
Gonsebatt ME, Vega L, Herrera LA, Montero R, Rojas E, et al. (1992). Inorganic arsenic effects on human lymphocyte stimulation and proliferation. Toxicol Appl Pharmacol. 245: 244-251.
-
(1992)
Toxicol Appl Pharmacol.
, vol.245
, pp. 244-251
-
-
Gonsebatt, M.E.1
Vega, L.2
Herrera, L.A.3
Montero, R.4
Rojas, E.5
-
7
-
-
84863845910
-
Inorganic arsenic impairs proliferation and cytokine expression in human primary lymphocytes
-
Morzadec C, Bouezzedine F, Macoch M, Fardel O, Vernhet L (2012). Inorganic arsenic impairs proliferation and cytokine expression in human primary lymphocytes. Toxicology 300: 46-56.
-
(2012)
Toxicology
, vol.300
, pp. 46-56
-
-
Morzadec, C.1
Bouezzedine, F.2
Macoch, M.3
Fardel, O.4
Vernhet, L.5
-
8
-
-
84863818175
-
Inorganic arsenic represses interleukin-17A expression in human activated th17 lymphocytes
-
Morzadec C, Macoch M, Robineau M, Sparfel L, Fardel O, et al. (2012). Inorganic arsenic represses interleukin-17A expression in human activated Th17 lymphocytes. Toxicol Appl Pharmacol 262: 217-222.
-
(2012)
Toxicol Appl Pharmacol
, vol.262
, pp. 217-222
-
-
Morzadec, C.1
Macoch, M.2
Robineau, M.3
Sparfel, L.4
Fardel, O.5
-
9
-
-
70350241787
-
Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate
-
Drobna Z, Naranmandura H, Kubachka KM, Edwards BC, Herbin-Davis K, et al. (2009). Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate. Chem Res Toxicol. 22: 1713-1720.
-
(2009)
Chem Res Toxicol.
, vol.22
, pp. 1713-1720
-
-
Drobna, Z.1
Naranmandura, H.2
Kubachka, K.M.3
Edwards, B.C.4
Herbin-Davis, K.5
-
10
-
-
84859189606
-
Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh
-
Pierce BL, Kibriya MG, Tong L, Jasmine F, Argos M, et al. (2012) Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh. PLoS Genet. 8: e1002522.
-
(2012)
PLoS Genet.
, vol.8
, pp. e1002522
-
-
Pierce, B.L.1
Kibriya, M.G.2
Tong, L.3
Jasmine, F.4
Argos, M.5
-
11
-
-
0030220509
-
Human T cells are highly sensitive to suppression of mitogenesis by polycyclic aromatic hydrocarbons and this effect is differentially reversed by alpha-naphthoflavone
-
Davila DR, Romero DL, Burchiel SW (1996). Human T cells are highly sensitive to suppression of mitogenesis by polycyclic aromatic hydrocarbons and this effect is differentially reversed by alpha-naphthoflavone. Toxicol Appl Pharmacol. 139: 333-341.
-
(1996)
Toxicol Appl Pharmacol
, vol.139
, pp. 333-341
-
-
Davila, D.R.1
Romero, D.L.2
Burchiel, S.W.3
-
12
-
-
0037348643
-
Development and application of a robust speciation method for determination of six arsenic compounds present in human urine
-
Milstein LS, Essader A, Pellizzari ED, Fernando RA, Raymer JH, et al. (2003). Development and application of a robust speciation method for determination of six arsenic compounds present in human urine. Environ Health Perspect. 111: 293-296.
-
(2003)
Environ Health Perspect.
, vol.111
, pp. 293-296
-
-
Milstein, L.S.1
Essader, A.2
Pellizzari, E.D.3
Fernando, R.A.4
Raymer, J.H.5
-
13
-
-
0015362658
-
Potentiation of the T-lymphocyte response to mitogens. I. The responding cell
-
Gery I, Gershon RK, Waksman BH (1972). Potentiation of the T-lymphocyte response to mitogens. I. The responding cell. J Exp Med. 136: 128-142.
-
(1972)
J Exp Med.
, vol.136
, pp. 128-142
-
-
Gery, I.1
Gershon, R.K.2
Waksman, B.H.3
-
14
-
-
85067750708
-
Arsenic alters monocyte superoxide anion and nitric oxide production in environmentally exposed children
-
Luna AL, Acosta-Saavedra LC, Lopez-Carrillo L, Conde P, Vera E, et al. (2010). Arsenic alters monocyte superoxide anion and nitric oxide production in environmentally exposed children. Mutation Research, 283: 91-95.
-
(2010)
Mutation Research
, vol.283
, pp. 91-95
-
-
Luna, A.L.1
Acosta-Saavedra, L.C.2
Lopez-Carrillo, L.3
Conde, P.4
Vera, E.5
-
15
-
-
0024553284
-
The anti-T cell monoclonal antibody 9.3 (anti-CD28) provides a helper signal and bypasses the need for accessory cells in T cell activation with immobilized anti-CD3 and mitogens
-
Baroja ML, Lorre K, Van Vaeck F, Ceuppens JL (1989). The anti-T cell monoclonal antibody 9.3 (anti-CD28) provides a helper signal and bypasses the need for accessory cells in T cell activation with immobilized anti-CD3 and mitogens. Cell Immunol. 120: 205-217.
-
(1989)
Cell Immunol.
, vol.120
, pp. 205-217
-
-
Baroja, M.L.1
Lorre, K.2
Van Vaeck, F.3
Ceuppens, J.L.4
-
16
-
-
69949096644
-
Low-dose arsenic compromises the immune system response to influenza A infection in vivo
-
Kozul CD, Ely KH, Enelow RI, Hamilton JW (2009). Low-dose arsenic compromises the immune system response to influenza A infection in vivo. Environ Health Perspect 117: 1441-1447.
-
(2009)
Environ Health Perspect
, vol.117
, pp. 1441-1447
-
-
Kozul, C.D.1
Ely, K.H.2
Enelow, R.I.3
Hamilton, J.W.4
-
17
-
-
33846482728
-
Mitogenic signal transduction caused by monomethylarsonous acid in human bladder cells: Role in arsenic-induced carcinogenesis
-
Eblin KE, Bredfeldt TG, Buffington S, Gandolfi AJ (2007). Mitogenic signal transduction caused by monomethylarsonous acid in human bladder cells: role in arsenic-induced carcinogenesis. Toxicol Sci. 95: 321-330.
-
(2007)
Toxicol Sci.
, vol.95
, pp. 321-330
-
-
Eblin, K.E.1
Bredfeldt, T.G.2
Buffington, S.3
Gandolfi, A.J.4
-
18
-
-
77955918808
-
Arsenic-induced cell proliferation is associated with enhanced ROS generation, erk signaling and cyclin A expression
-
Chowdhury R, Chatterjee R, Giri AK, Mandal C, Chaudhuri K (2010). Arsenic-induced cell proliferation is associated with enhanced ROS generation, Erk signaling and Cyclin A expression. Toxicol Lett. 198: 263-71.
-
(2010)
Toxicol Lett.
, vol.198
, pp. 263-271
-
-
Chowdhury, R.1
Chatterjee, R.2
Giri, A.K.3
Mandal, C.4
Chaudhuri, K.5
-
19
-
-
45049083520
-
Arsenic interferes with the signaling transduction pathway of T cell receptor activation by increasing basal and induced phosphorylation of lck and fyn in spleen cells
-
Soto-Peña GA, Vega L (2008). Arsenic interferes with the signaling transduction pathway of T cell receptor activation by increasing basal and induced phosphorylation of Lck and Fyn in spleen cells. Toxicol Appl Pharmacol. 230: 216-226.
-
(2008)
Toxicol Appl Pharmacol
, vol.230
, pp. 216-226
-
-
Soto-Peña, G.A.1
Vega, L.2
-
20
-
-
33746309999
-
Blood arsenic as a biomarker of arsenic exposure: Results from a prospective study
-
Hall M, Chen Y, Ahsan H, Slavkovich V, A van Geen, et al. (2006). Blood arsenic as a biomarker of arsenic exposure: results from a prospective study. Toxicology. 225: 225-233.
-
(2006)
Toxicology
, vol.225
, pp. 225-233
-
-
Hall, M.1
Chen, Y.2
Ahsan, H.3
Slavkovich, V.4
Van Geen, A.5
-
21
-
-
79955867370
-
Polymorphisms in arsenic (+III oxidation state) methyltransferase (AS3MT) predict gene expression of AS3MT as well as arsenic metabolism
-
Engström K, Vahter M, Mlakar SJ, Concha G, Nermell B, et al. (2011) Polymorphisms in arsenic (+III oxidation state) methyltransferase (AS3MT) predict gene expression of AS3MT as well as arsenic metabolism. Environ Health Perspect. 119: 182-188.
-
(2011)
Environ Health Perspect
, vol.119
, pp. 182-188
-
-
Engström, K.1
Vahter, M.2
Mlakar, S.J.3
Concha, G.4
Nermell, B.5
-
22
-
-
84892468257
-
Arsenic metabolism efficiency has a causal role in arsenic toxicity: Mendelian randomization and gene-environment interaction
-
Pierce BL, Tong L, Argos M, Gao J, Farzana J, et al. (2013). Arsenic metabolism efficiency has a causal role in arsenic toxicity: Mendelian randomization and gene-environment interaction. Int J Epidemiol. 42: 1862-1871. doi: 10.1093/ije/dyt182.
-
(2013)
Int J Epidemiol.
, vol.42
, pp. 1862-1871
-
-
Pierce, B.L.1
Tong, L.2
Argos, M.3
Gao, J.4
Farzana, J.5
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