Of mice and men: Modeling cardiovascular complexity in diabetes. Focus on “Mitochondrial inefficiencies and anoxic ATP hydrolysis capacities in diabetic rat heart”

American Journal of Physiology - Cell Physiology

Volumn 307, Issue 6, 2014, Pages C497-C498

  Patel, Hemal H ^a,b   McDonough, Alicia A ^c  

^a VA SAN DIEGO HEALTHCARE SYSTEM   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF SOUTHERN CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; REACTIVE OXYGEN METABOLITE;

DIABETIC CARDIOMYOPATHY; EDITORIAL; ENZYME SYNTHESIS; GENETIC MODEL; HEART LEFT VENTRICLE ENDDIASTOLIC PRESSURE; HEART MITOCHONDRION; HEART MUSCLE; HEART MUSCLE ISCHEMIA; HUMAN; HYDROLYSIS; IMMUNE RESPONSE; INSULIN DEPENDENT DIABETES MELLITUS; METABOLIC DISORDER; MITOCHONDRIAL MEMBRANE POTENTIAL; NONHUMAN; STREPTOZOTOCIN-INDUCED DIABETES MELLITUS; TISSUE HOMOGENATE; ANIMAL; ANOXIA; ENERGY METABOLISM; MALE; METABOLISM; NOTE;

ADENOSINE TRIPHOSPHATE; ANIMALS; ANOXIA; DIABETIC CARDIOMYOPATHIES; ENERGY METABOLISM; MALE; MITOCHONDRIA, HEART; MYOCARDIUM;

EID: 84907154001     PISSN: 03636143     EISSN: 15221563     Source Type: Journal    
DOI: 10.1152/ajpcell.00241.2014     Document Type: Editorial

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