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Volumn 10, Issue 1, 2013, Pages 73-79
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Mismatch repair deficient mice show susceptibility to oxidative stress-induced intestinal carcinogenesis.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
BETA CATENIN;
BROMATE;
CATNB PROTEIN, MOUSE;
MSH2 PROTEIN, MOUSE;
POTASSIUM BROMATE;
PRIMER DNA;
PROTEIN MSH2;
ANIMAL;
APOPTOSIS;
ARTICLE;
BASE MISPAIRING;
CARCINOGENESIS;
CELL DEATH;
CHEMICALLY INDUCED DISORDER;
DNA REPAIR;
DRUG EFFECT;
GENETICS;
HEREDITARY NONPOLYPOSIS COLORECTAL CANCER;
INTESTINE TUMOR;
MOUSE;
MUTAGENESIS;
MUTATION;
NICK END LABELING;
NUCLEOTIDE SEQUENCE;
OXIDATIVE DNA DAMAGE;
OXIDATIVE STRESS;
PATHOLOGY;
REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION;
SMALL INTESTINE;
WNT SIGNALING PATHWAY;
CELL DEATH;
HNPCC;
MUTAGENESIS;
OXIDATIVE DNA DAMAGE;
WNT SIGNALING PATHWAY;
ANIMALS;
APOPTOSIS;
BASE PAIR MISMATCH;
BASE SEQUENCE;
BETA CATENIN;
BROMATES;
CARCINOGENESIS;
DNA PRIMERS;
DNA REPAIR;
IN SITU NICK-END LABELING;
INTESTINAL NEOPLASMS;
INTESTINE, SMALL;
MICE;
MUTATION;
MUTS HOMOLOG 2 PROTEIN;
OXIDATIVE STRESS;
REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;
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EID: 84906552394
PISSN: None
EISSN: 14492288
Source Type: Journal
DOI: 10.7150/ijbs.5750 Document Type: Article |
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Times cited : (23)
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References (0)
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