Overexpression of junctophilin-2 does not enhance baseline function but attenuates heart failure development after cardiac stress

Proceedings of the National Academy of Sciences of the United States of America

Volumn 111, Issue 33, 2014, Pages 12240-12245

  Guo, Ang ^a   Zhang, Xiaoying ^b   Iyer, Venkat Ramesh ^c   Chen, Biyi ^a   Zhang, Caimei ^a   Kutschke, William J ^a   Weiss, Robert M ^a   Franzini Armstrong, Clara ^d   Song, Long Sheng ^a  

^a UNIVERSITY OF IOWA   (United States)

^b TEMPLE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c CHILDREN S HOSPITAL OF PHILADELPHIA   (United States)

^d UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

Cardiac dyads; Electron microscopy

Indexed keywords

CALCIUM; JUNCTOPHILIN 2; MEMBRANE PROTEIN; SODIUM CALCIUM EXCHANGE PROTEIN; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ARTICLE; DISORIENTATION; DOWN REGULATION; EXCITATION CONTRACTION COUPLING; HEART FAILURE; HEART FUNCTION; HEART HYPERTROPHY; HEART LEFT VENTRICLE; HEART LEFT VENTRICLE OVERLOAD; HEART PROTECTION; HEART RIGHT VENTRICLE; HEART STRESS; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SURVIVAL; TRANSGENIC MOUSE; TRANSVERSE TUBULAR SYSTEM;

CARDIAC DYADS; ELECTRON MICROSCOPY; IN SITU CA2+ IMAGING;

ANIMALS; CALCIUM; HEART FAILURE; MEMBRANE PROTEINS; MICE; MICE, TRANSGENIC; MUSCLE PROTEINS; MYOCYTES, CARDIAC; STRESS, PHYSIOLOGICAL; VENTRICULAR PRESSURE;

EID: 84906319223     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1412729111     Document Type: Article

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