Glia are critical for the neuropathology of complex I deficiency in Drosophila

Human Molecular Genetics

Volumn 23, Issue 17, 2014, Pages 4686-4692

  Hegde, Vijay R ^a   Vogel, Rutger ^b,c   Feany, Mel B ^a  

^a BRIGHAM AND WOMEN S HOSPITAL   (United States)

^b HARVARD MEDICAL SCHOOL   (United States)

^c RADBOUD UNIVERSITY MEDICAL CENTER   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

EXCITATORY AMINO ACID TRANSPORTER 1; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE 1; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); TRANSCRIPTION FACTOR GAL4; NEUROTOXIN;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN DEGENERATION; CELL LOSS; CLINICAL FEATURE; COMPLEX I DEFICIENCY; CONTROLLED STUDY; DROSOPHILA; GENE SILENCING; GLIA; GRAY MATTER; MITOCHONDRIAL RESPIRATION; MOTOR DYSFUNCTION; NERVE CELL; NERVE CELL NECROSIS; NEUROPATHOLOGY; NEUROTOXICITY; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; RESPIRATORY CHAIN; WHITE MATTER; ANIMAL; DEFICIENCY; DROSOPHILA MELANOGASTER; DRUG EFFECTS; METABOLISM; MITOCHONDRION; PATHOLOGY; ULTRASTRUCTURE;

ANIMALS; DROSOPHILA MELANOGASTER; ELECTRON TRANSPORT COMPLEX I; GENE KNOCKDOWN TECHNIQUES; MITOCHONDRIA; NEUROGLIA; NEUROTOXINS; PHENOTYPE;

EID: 84905637827     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/ddu188     Document Type: Article

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