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Volumn 278, Issue 2, 2014, Pages 100-106
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The inhibition of human T cell proliferation by the caspase inhibitor z-VAD-FMK is mediated through oxidative stress.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
BENZYLOXYCARBONYLVALYL ALANYL ASPARTYL FLUOROMETHYL KETONE;
BENZYLOXYCARBONYLVALYL-ALANYL-ASPARTYL FLUOROMETHYL KETONE;
CASPASE;
CASPASE INHIBITOR;
GLUTATHIONE;
GROWTH INHIBITOR;
PEPTIDE CHLOROMETHYL KETONE;
REACTIVE OXYGEN METABOLITE;
ARTICLE;
CELL CULTURE;
CELL PROLIFERATION;
DOSE RESPONSE;
DRUG EFFECT;
HUMAN;
IMMUNOLOGY;
LYMPHOCYTE ACTIVATION;
METABOLISM;
MONONUCLEAR CELL;
OXIDATIVE STRESS;
PHYSIOLOGY;
T LYMPHOCYTE ACTIVATION;
T LYMPHOCYTE SUBPOPULATION;
T LYMPHOCYTES;
THIOLS;
Z-VAD-FMK;
CASPASES;
OXIDATIVE STRESS;
T CELL ACTIVATION;
T LYMPHOCYTES;
THIOLS;
Z-VAD-FMK;
AMINO ACID CHLOROMETHYL KETONES;
CASPASE INHIBITORS;
CELL PROLIFERATION;
CELLS, CULTURED;
DOSE-RESPONSE RELATIONSHIP, IMMUNOLOGIC;
GLUTATHIONE;
GROWTH INHIBITORS;
HUMANS;
LEUKOCYTES, MONONUCLEAR;
LYMPHOCYTE ACTIVATION;
OXIDATIVE STRESS;
REACTIVE OXYGEN SPECIES;
T-LYMPHOCYTE SUBSETS;
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EID: 84905403182
PISSN: None
EISSN: 10960333
Source Type: Journal
DOI: 10.1016/j.taap.2014.04.014 Document Type: Article |
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Times cited : (15)
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References (0)
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