Spatial memory deficits in a mouse model of late-onset Alzheimer's disease are caused by zinc supplementation and correlate with amyloid-beta levels

Frontiers in Aging Neuroscience

Volumn 6, Issue JUL, 2014, Pages

  Flinn, Jane M ^a   Lorenzo Bozzelli, P ^a   Adlard, Paul A ^b   Railey, Angela M ^a  

^a GEORGE MASON UNIVERSITY   (United States)

^b UNIVERSITY OF MELBOURNE   (Australia)

Author keywords

Apolipoprotein E; Barnes maze; Copper; CRND8; HAPP; Metals; Transgenic mice; ZnT3

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; APOLIPOPROTEIN E4; GLIAL FIBRILLARY ACIDIC PROTEIN; TAP WATER; UNCLASSIFIED DRUG; ZINC; ZINC TRANSPORTER; ZINC TRANSPORTER 3;

ALZHEIMER DISEASE; AMNESIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BARNES MAZE TEST; BRAIN TISSUE; CONTROLLED STUDY; DIET SUPPLEMENTATION; FEMALE; GENE MUTATION; GENOTYPE; HABITUATION; HETEROZYGOTE; HUMAN; LATENT PERIOD; MALE; MOUSE; NONHUMAN; PROTEIN ANALYSIS; SEX DIFFERENCE; SPATIAL MEMORY; WESTERN BLOTTING; APOLIPOPROTEIN E GENE; BINDING AFFINITY; CORRELATION ANALYSIS; GENE; GENE INTERACTION; MINERAL SUPPLEMENTATION; PROTEIN INTERACTION; RISK ASSESSMENT;

EID: 84904708745     PISSN: None     EISSN: 16634365     Source Type: Journal    
DOI: 10.3389/fnagi.2014.00174     Document Type: Article

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