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Volumn 3, Issue 2, 2014, Pages
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CCR5 inhibition prevents cardiac dysfunction in the SIV/macaque model of HIV.
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Author keywords
[No Author keywords available]
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Indexed keywords
CHEMOKINE;
CHEMOKINE RECEPTOR CCR5;
CYCLOHEXANE DERIVATIVE;
HUMAN IMMUNODEFICIENCY VIRUS FUSION INHIBITOR;
LIGAND;
MARAVIROC;
TRIAZOLE DERIVATIVE;
ANIMAL;
ANIMAL MODEL;
ARTICLE;
DIASTOLE;
DIASTOLIC FUNCTION;
DISEASE MODEL;
DOPPLER ECHOCARDIOGRAPHY;
DRUG ANTAGONISM;
DRUG EFFECT;
ECHOCARDIOGRAPHY;
HEART MUSCLE CELL;
HEART VENTRICLE;
HEART VENTRICLE FUNCTION;
HUMAN IMMUNODEFICIENCY VIRUS;
METABOLISM;
PATHOPHYSIOLOGY;
RHESUS MONKEY;
SIGNAL TRANSDUCTION;
SIMIAN ACQUIRED IMMUNODEFICIENCY SYNDROME;
VIROLOGY;
VIRUS LOAD;
ANIMAL MODEL;
CHEMOKINE;
DIASTOLIC FUNCTION;
ECHOCARDIOGRAPHY;
HIV;
ANIMALS;
CYCLOHEXANES;
DIASTOLE;
DISEASE MODELS, ANIMAL;
ECHOCARDIOGRAPHY, DOPPLER;
HEART VENTRICLES;
HIV FUSION INHIBITORS;
LIGANDS;
MACACA MULATTA;
MYOCYTES, CARDIAC;
RECEPTORS, CCR5;
SIGNAL TRANSDUCTION;
SIMIAN ACQUIRED IMMUNODEFICIENCY SYNDROME;
TRIAZOLES;
VENTRICULAR DYSFUNCTION;
VENTRICULAR FUNCTION;
VIRAL LOAD;
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EID: 84904639892
PISSN: None
EISSN: 20479980
Source Type: Journal
DOI: 10.1161/JAHA.114.000874 Document Type: Article |
Times cited : (27)
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References (0)
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