-
1
-
-
84959777555
-
Ileocolonoscopic findings in seronegative spondylarthropathies
-
Mielants H, Veys EM, Cuvelier C, et al. Ileocolonoscopic findings in seronegative spondylarthropathies. Br J Rheumatol 1988;27:95-105.
-
(1988)
Br J Rheumatol
, vol.27
, pp. 95-105
-
-
Mielants, H.1
Veys, E.M.2
Cuvelier, C.3
-
2
-
-
0028848441
-
The evolution of spondyloarthropathies in relation to gut histology. I. Clinical aspects
-
Mielants H, Veys EM, De Vos M, et al. The evolution of spondyloarthropathies in relation to gut histology. I. Clinical aspects. J Rheumatol 1995;22:2266-72.
-
(1995)
J Rheumatol
, vol.22
, pp. 2266-2272
-
-
Mielants, H.1
Veys, E.M.2
De Vos, M.3
-
3
-
-
0028823453
-
The evolution of spondyloarthropathies in relation to gut histology. II. Histological aspects
-
Mielants H, Veys EM, Cuvelier C, et al. The evolution of spondyloarthropathies in relation to gut histology. II. Histological aspects. J Rheumatol 1995;22:2273-8.
-
(1995)
J Rheumatol
, vol.22
, pp. 2273-2278
-
-
Mielants, H.1
Veys, E.M.2
Cuvelier, C.3
-
4
-
-
0141835046
-
Constitutive p40 promoter activation and IL-23 production in the terminal ileum mediated by dendritic cells
-
DOI 10.1172/JCI200317464
-
Becker C, Wirtz S, Blessing M, et al. Constitutive p40 promoter activation and IL-23 production in the terminal ileum mediated by dendritic cells. J Clin Invest 2003;112:693-706. (Pubitemid 38057710)
-
(2003)
Journal of Clinical Investigation
, vol.112
, Issue.5
, pp. 693-706
-
-
Becker, C.1
Wirtz, S.2
Blessing, M.3
Pirhonen, J.4
Strand, D.5
Bechthold, O.6
Frick, J.7
Galle, P.R.8
Autenrieth, I.9
Neurath, M.F.10
-
5
-
-
65249152431
-
Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis
-
Ciccia F, Bombardieri M, Principato A, et al. Overexpression of interleukin-23, but not interleukin-17, as an immunologic signature of subclinical intestinal inflammation in ankylosing spondylitis. Arthritis Rheum 2009;60:955-65.
-
(2009)
Arthritis Rheum
, vol.60
, pp. 955-965
-
-
Ciccia, F.1
Bombardieri, M.2
Principato, A.3
-
6
-
-
42449146934
-
Association of interleukin-23 receptor variants with ankylosing spondylitis
-
DOI 10.1002/art.23389
-
Rahman P, Inman RD, Gladman DD, et al. Association of interleukin-23 receptor variants with ankylosing spondylitis. Arthritis Rheum 2008;58:1020-5. (Pubitemid 351564003)
-
(2008)
Arthritis and Rheumatism
, vol.58
, Issue.4
, pp. 1020-1025
-
-
Rahman, P.1
Inman, R.D.2
Gladman, D.D.3
Reeve, J.P.4
Peddle, L.5
Maksymowych, W.P.6
-
7
-
-
53049097091
-
The IL23R Arg381Gln non-synonymous polymorphism confers susceptibility to ankylosing spondylitis
-
Rueda B, Orozco G, Raya E, et al. The IL23R Arg381Gln non-synonymous polymorphism confers susceptibility to ankylosing spondylitis. Ann Rheum Dis 2008;67:1451-4.
-
(2008)
Ann Rheum Dis
, vol.67
, pp. 1451-1454
-
-
Rueda, B.1
Orozco, G.2
Raya, E.3
-
8
-
-
75749085686
-
Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci
-
Reveille JD, Sims AM, Danoy P, et al. Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci. Nat Genet 2010;42:123-7.
-
(2010)
Nat Genet
, vol.42
, pp. 123-127
-
-
Reveille, J.D.1
Sims, A.M.2
Danoy, P.3
-
9
-
-
82455210509
-
Ankylosing spondylitis macrophage production of higher levels of interleukin-23 in response to lipopolysaccharide without induction of a significant unfolded protein response
-
Zeng L, Lindstrom MJ, Smith JA. Ankylosing spondylitis macrophage production of higher levels of interleukin-23 in response to lipopolysaccharide without induction of a significant unfolded protein response. Arthritis Rheum 2011;63:3807-17.
-
(2011)
Arthritis Rheum
, vol.63
, pp. 3807-3817
-
-
Zeng, L.1
Lindstrom, M.J.2
Smith, J.A.3
-
10
-
-
79954450170
-
Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis
-
Mei Y, Pan F, Gao J, et al. Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis. Clin Rheumatol 2011;30:269-73.
-
(2011)
Clin Rheumatol
, vol.30
, pp. 269-273
-
-
Mei, Y.1
Pan, F.2
Gao, J.3
-
11
-
-
77949444874
-
Systemic levels of IL-23 are strongly associated with disease activity in rheumatoid arthritis but not spondyloarthritis
-
Melis L, Vandooren B, Kruithof E, et al. Systemic levels of IL-23 are strongly associated with disease activity in rheumatoid arthritis but not spondyloarthritis. Ann Rheum Dis 2010;69:618-23.
-
(2010)
Ann Rheum Dis
, vol.69
, pp. 618-623
-
-
Melis, L.1
Vandooren, B.2
Kruithof, E.3
-
12
-
-
69449086287
-
HLA-B27 misfolding and the unfolded protein response augment interleukin-23 production and are associated with Th17 activation in transgenic rats
-
DeLay ML, Turner MJ, Klenk EI, et al. HLA-B27 misfolding and the unfolded protein response augment interleukin-23 production and are associated with Th17 activation in transgenic rats. Arthritis Rheum 2009;60:2633-43.
-
(2009)
Arthritis Rheum
, vol.60
, pp. 2633-2643
-
-
Delay, M.L.1
Turner, M.J.2
Klenk, E.I.3
-
13
-
-
84857444876
-
Interleukin 23 production by intestinal CD103 (+)CD11b (+) dendritic cells in response to bacterial flagellin enhances mucosal innate immune defense
-
Kinnebrew MA, Buffie CG, Diehl GE, et al. Interleukin 23 production by intestinal CD103 (+)CD11b (+) dendritic cells in response to bacterial flagellin enhances mucosal innate immune defense. Immunity 2012;36:276-87.
-
(2012)
Immunity
, vol.36
, pp. 276-287
-
-
Kinnebrew, M.A.1
Buffie, C.G.2
Diehl, G.E.3
-
14
-
-
78049271424
-
Endoplasmic reticulum stress-induced transcription factor, CHOP, is crucial for dendritic cell IL-23 expression
-
Goodall JC, Wu C, Zhang Y, et al. Endoplasmic reticulum stress-induced transcription factor, CHOP, is crucial for dendritic cell IL-23 expression. Proc Natl Acad Sci USA 2010;107:17698-703.
-
(2010)
Proc Natl Acad Sci USA
, vol.107
, pp. 17698-17703
-
-
Goodall, J.C.1
Wu, C.2
Zhang, Y.3
-
15
-
-
83655191565
-
Epithelial barrier: An interface for the cross-communication between gut flora and immune system
-
Goto Y, Kiyono H. Epithelial barrier: an interface for the cross-communication between gut flora and immune system. Immunol Rev 2012;245:147-63.
-
(2012)
Immunol Rev
, vol.245
, pp. 147-163
-
-
Goto, Y.1
Kiyono, H.2
-
16
-
-
84875892111
-
Autophagy as a stress-response and quality-control mechanism: Implications for cell injury and human disease
-
Murrow L, Debnath J. Autophagy as a stress-response and quality-control mechanism: implications for cell injury and human disease. Annu Rev Pathol 2013;8:105-37.
-
(2013)
Annu Rev Pathol
, vol.8
, pp. 105-137
-
-
Murrow, L.1
Debnath, J.2
-
17
-
-
41949101594
-
Toll-like receptors control autophagy
-
DOI 10.1038/emboj.2008.31, PII EMBOJ200831
-
Delgado MA, Elmaoued RA, Davis AS, et al. Toll-like receptors control autophagy. EMBO J 2008;27:1110-21. (Pubitemid 351508141)
-
(2008)
EMBO Journal
, vol.27
, Issue.7
, pp. 1110-1121
-
-
Delgado, M.A.1
Elmaoued, R.A.2
Davis, A.S.3
Kyei, G.4
Deretic, V.5
-
18
-
-
37549043217
-
Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosis
-
Sanjuan MA, Dillon CP, Tait SW, et al. Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosis. Nature 2007;450:1253-7.
-
(2007)
Nature
, vol.450
, pp. 1253-1257
-
-
Sanjuan, M.A.1
Dillon, C.P.2
Tait, S.W.3
-
19
-
-
53549117440
-
Induction of autophagy via innate bacterial recognition
-
Yano T, Kurata S. Induction of autophagy via innate bacterial recognition. Autophagy 2008;4:958-60.
-
(2008)
Autophagy
, vol.4
, pp. 958-960
-
-
Yano, T.1
Kurata, S.2
-
20
-
-
79955111850
-
Hepatic autophagy mediates endoplasmic reticulum stress-induced degradation of misfolded apolipoprotein B
-
Qiu W, Zhang J, Dekker MJ, et al. Hepatic autophagy mediates endoplasmic reticulum stress-induced degradation of misfolded apolipoprotein B. Hepatology 2011;53:1515-25.
-
(2011)
Hepatology
, vol.53
, pp. 1515-1525
-
-
Qiu, W.1
Zhang, J.2
Dekker, M.J.3
-
22
-
-
84856111924
-
The unfolded protein response: Controlling cell fate decisions under ER stress and beyond
-
Hetz C. The unfolded protein response: controlling cell fate decisions under ER stress and beyond. Nat Rev Mol Cell Biol 2012;13:89-102.
-
(2012)
Nat Rev Mol Cell Biol
, vol.13
, pp. 89-102
-
-
Hetz, C.1
-
23
-
-
33751549487
-
Paneth cells: Leukocyte-like mediators of innate immunity in the intestine
-
DOI 10.1189/jlb.1005556
-
Keshav S. Paneth cells: leukocyte-like mediators of innate immunity in the intestine. J Leukoc Biol 2006;80:500-8. (Pubitemid 44835783)
-
(2006)
Journal of Leukocyte Biology
, vol.80
, Issue.3
, pp. 500-508
-
-
Keshav, S.1
-
24
-
-
84861796578
-
Interleukin-22 and IL-22-producing NKp44 (+) NK cells in the subclinical gut inflammation of patients with ankylosing spondylitis
-
Ciccia F, Accardo-Palumbo A, Alessandro R, et al. Interleukin-22 and IL-22-producing NKp44 (+) NK cells in the subclinical gut inflammation of patients with ankylosing spondylitis. Arthritis Rheum 2012;64:1869-78.
-
(2012)
Arthritis Rheum
, vol.64
, pp. 1869-1878
-
-
Ciccia, F.1
Accardo-Palumbo, A.2
Alessandro, R.3
-
25
-
-
0022533999
-
Monoclonal antibodies raised against denatured HLA-B locus heavy chains permit biochemical characterization of certain HLA-C locus products
-
Stam NJ, Spits H, Ploegh HL. Monoclonal antibodies raised against denatured HLA-B locus heavy chains permit biochemical characterization of certain HLA-C locus products. J Immunol 1986;137:2299-306. (Pubitemid 16020728)
-
(1986)
Journal of Immunology
, vol.137
, Issue.7
, pp. 2299-2306
-
-
Stam, N.J.1
Spits, H.2
Ploegh, H.L.3
-
26
-
-
78650072547
-
Expansion of intestinal CD4+CD25 (high) Treg cells in patients with ankylosing spondylitis: A putative role for interleukin-10 in preventing intestinal Th17 response
-
Ciccia F, Accardo-Palumbo A, Giardina A, et al. Expansion of intestinal CD4+CD25 (high) Treg cells in patients with ankylosing spondylitis: a putative role for interleukin-10 in preventing intestinal Th17 response. Arthritis Rheum 2010;62:3625-34.
-
(2010)
Arthritis Rheum
, vol.62
, pp. 3625-3634
-
-
Ciccia, F.1
Accardo-Palumbo, A.2
Giardina, A.3
-
27
-
-
0035947778
-
Feedback inhibition of the unfolded protein response by GADD34-mediated dephosphorylation of eIF2alpha
-
Novoa I, Zeng H, Harding HP, et al. Feedback inhibition of the unfolded protein response by GADD34-mediated dephosphorylation of eIF2alpha. J Cell Biol 2001;153:1011-22.
-
(2001)
J Cell Biol
, vol.153
, pp. 1011-1022
-
-
Novoa, I.1
Zeng, H.2
Harding, H.P.3
-
29
-
-
79952133558
-
HRD1 and UBE2J1 target misfolded MHC class i heavy chains for endoplasmic reticulum-associated degradation
-
Burr ML, Cano F, Svobodova S, et al. HRD1 and UBE2J1 target misfolded MHC class I heavy chains for endoplasmic reticulum-associated degradation. Proc Natl Acad Sci USA 2011;108:2034-9.
-
(2011)
Proc Natl Acad Sci USA
, vol.108
, pp. 2034-2039
-
-
Burr, M.L.1
Cano, F.2
Svobodova, S.3
-
31
-
-
23444431837
-
HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response
-
Turner MJ, Sowders DP, DeLay ML, et al. HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response. J Immunol 2005;175:2438-48. (Pubitemid 41113854)
-
(2005)
Journal of Immunology
, vol.175
, Issue.4
, pp. 2438-2448
-
-
Turner, M.J.1
Sowders, D.P.2
DeLay, M.L.3
Mohapatra, R.4
Bai, S.5
Smith, J.A.6
Brandewie, J.R.7
Taurog, J.D.8
Colbert, R.A.9
-
32
-
-
56249135538
-
A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells
-
Cadwell K, Liu JY, Brown SL, et al. A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells. Nature 2008;456:259-63.
-
(2008)
Nature
, vol.456
, pp. 259-263
-
-
Cadwell, K.1
Liu, J.Y.2
Brown, S.L.3
-
33
-
-
84864318195
-
Chaperone-mediated autophagy: A unique way to enter the lysosome world
-
Kaushik S, Cuervo AM. Chaperone-mediated autophagy: a unique way to enter the lysosome world. Trends Cell Biol 2012;22:407-17.
-
(2012)
Trends Cell Biol
, vol.22
, pp. 407-417
-
-
Kaushik, S.1
Cuervo, A.M.2
-
34
-
-
84860136921
-
Abnormal activation of autophagy-induced crinophagy in Paneth cells from patients with Crohn's disease
-
Thachil E, Hugot JP, Arbeille B, et al. Abnormal activation of autophagy-induced crinophagy in Paneth cells from patients with Crohn's disease. Gastroenterology 2012;142:1097-9.
-
(2012)
Gastroenterology
, vol.142
, pp. 1097-1099
-
-
Thachil, E.1
Hugot, J.P.2
Arbeille, B.3
-
35
-
-
84862295360
-
Guidelines for the use and interpretation of assays for monitoring autophagy
-
Klionsky DJ, Abdalla FC, Abeliovich H, et al. Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy 2012;8:445-544.
-
(2012)
Autophagy
, vol.8
, pp. 445-544
-
-
Klionsky, D.J.1
Abdalla, F.C.2
Abeliovich, H.3
-
36
-
-
78649354905
-
Over-expression of paneth cell-derived anti-microbial peptides in the gut of patients with ankylosing spondylitis and subclinical intestinal inflammation
-
Ciccia F, Bombardieri M, Rizzo A, et al. Over-expression of paneth cell-derived anti-microbial peptides in the gut of patients with ankylosing spondylitis and subclinical intestinal inflammation. Rheumatology (Oxford) 2010;49:2076-83.
-
(2010)
Rheumatology (Oxford)
, vol.49
, pp. 2076-2083
-
-
Ciccia, F.1
Bombardieri, M.2
Rizzo, A.3
-
37
-
-
84867284676
-
Autophagy regulates IL-23 secretion and innate T cell responses through effects on IL-1 secretion
-
Peral de Castro C, Jones SA, Ní Cheallaigh C, et al. Autophagy regulates IL-23 secretion and innate T cell responses through effects on IL-1 secretion. J Immunol 2012;189:4144-53.
-
(2012)
J Immunol
, vol.189
, pp. 4144-4153
-
-
Peral De Castro, C.1
Jones, S.A.2
Ní Cheallaigh, C.3
-
39
-
-
33846211417
-
ER stress (PERK/eIF2α phosphorylation) mediates the polyglutamine-induced LC3 conversion, an essential step for autophagy formation
-
DOI 10.1038/sj.cdd.4401984, PII 4401984
-
Kouroku Y, Fujita E, Tanida I, et al. ER stress (PERK/eIF2alpha phosphorylation) mediates the polyglutamine-induced LC3 conversion, an essential step for autophagy formation. Cell Death Differ 2007;14:230-9. (Pubitemid 46092226)
-
(2007)
Cell Death and Differentiation
, vol.14
, Issue.2
, pp. 230-239
-
-
Kouroku, Y.1
Fujita, E.2
Tanida, I.3
Ueno, T.4
Isoai, A.5
Kumagai, H.6
Ogawa, S.7
Kaufman, R.J.8
Kominami, E.9
Momoi, T.10
-
41
-
-
66349112572
-
Autophagic elimination of misfolded procollagen aggregates in the endoplasmic reticulum as a means of cell protection
-
Ishida Y, Yamamoto A, Kitamura A, et al. Autophagic elimination of misfolded procollagen aggregates in the endoplasmic reticulum as a means of cell protection. Mol Biol Cell 2009;20:2744-54.
-
(2009)
Mol Biol Cell
, vol.20
, pp. 2744-2754
-
-
Ishida, Y.1
Yamamoto, A.2
Kitamura, A.3
-
42
-
-
80053104784
-
Role of chaperone mediated autophagy (CMA) in the degradation of misfolded N-CoR protein in non-small cell lung cancer (NSCLC) cells
-
Ali AB, Nin DS, Tam J, et al. Role of chaperone mediated autophagy (CMA) in the degradation of misfolded N-CoR protein in non-small cell lung cancer (NSCLC) cells. PLoS ONE 2011;6:e25268.
-
(2011)
PLoS ONE
, vol.6
-
-
Ali, A.B.1
Nin, D.S.2
Tam, J.3
|