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Volumn 10, Issue 6, 2014, Pages 1141-1142
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Deficient autophagy unravels the ROS paradox in chronic granulomatous disease
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Author keywords
Aspergillosis; CGD; Colitis; Crohn disease; Inflammation; Interleukin 1; LC3 associated phagocytosis
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Indexed keywords
CORTICOSTEROID;
INTERLEUKIN 1BETA;
REACTIVE OXYGEN METABOLITE;
RECOMBINANT INTERLEUKIN 1 RECEPTOR BLOCKING AGENT;
REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE;
REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;
ANTIINFLAMMATORY AGENT;
INTERLEUKIN 1 RECEPTOR;
INTERLEUKIN 1 RECEPTOR BLOCKING AGENT;
ALTERNATIVE MEDICINE;
ARTICLE;
ASPERGILLUS FUMIGATUS;
AUTOINFLAMMATORY DISEASE;
AUTOPHAGY;
CHRONIC GRANULOMATOUS DISEASE;
CROHN DISEASE;
CYTOKINE PRODUCTION;
GROWTH RETARDATION;
HUMAN;
INFECTIOUS COMPLICATION;
LIVER ABSCESS;
LUNG ASPERGILLOSIS;
MONOCYTE;
NONHUMAN;
PATHOGENESIS;
PERITONEUM MACROPHAGE;
PHAGOSOME;
STAPHYLOCOCCUS AUREUS;
ANIMAL;
ANTAGONISTS AND INHIBITORS;
COLITIS;
GRANULOMATOUS DISEASE, CHRONIC;
METABOLISM;
ANIMALS;
ANTI-INFLAMMATORY AGENTS;
COLITIS;
GRANULOMATOUS DISEASE, CHRONIC;
HUMANS;
INTERLEUKIN 1 RECEPTOR ANTAGONIST PROTEIN;
PHAGOSOMES;
RECEPTORS, INTERLEUKIN-1;
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EID: 84903577067
PISSN: 15548627
EISSN: 15548635
Source Type: Journal
DOI: 10.4161/auto.28638 Document Type: Article |
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Times cited : (34)
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References (0)
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