Rad GTPase deletion increases L-type calcium channel current leading to increased cardiac contraction.

Journal of the American Heart Association

Volumn 2, Issue 6, 2013, Pages

  Manning, Janet R ^a   Yin, Guo ^b   Kaminski, Catherine N ^b   Magyar, Janos ^b   Feng, Han Zhong ^b   Penn, John ^b   Sievert, Gail ^b   Thompson, Katherine ^b   Jin, J P ^b   Andres, Douglas A ^b   Satin, Jonathan ^b  

^a UNIVERSITY OF KENTUCKY   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

BETA ADRENERGIC RECEPTOR STIMULATING AGENT; CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE; CALCIUM CHANNEL L TYPE; CARDIOTONIC AGENT; RAS PROTEIN; RRAD PROTEIN, MOUSE;

129 MOUSE; ACTION POTENTIAL; ANIMAL; ARTICLE; C57BL MOUSE; DRUG EFFECT; ENZYMOLOGY; EXCITATION CONTRACTION COUPLING; GENE DELETION; GENETICS; GENOTYPE; HEART CONTRACTION; HEART MUSCLE CELL; HEART PACING; HEART RATE; HEART VENTRICLE PRESSURE; INOTROPY; KINETICS; KNOCKOUT MOUSE; L-TYPE CALCIUM CURRENT; METABOLISM; MOUSE; PHENOTYPE; RAD; RGK; SARCOMERE; UPREGULATION; Β-ADRENERGIC STIMULATION;

INOTROPY; L-TYPE CALCIUM CURRENT; RAD; RGK; Β-ADRENERGIC STIMULATION;

ACTION POTENTIALS; ADRENERGIC BETA-AGONISTS; ANIMALS; CALCIUM CHANNELS, L-TYPE; CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASES; CARDIAC PACING, ARTIFICIAL; CARDIOTONIC AGENTS; EXCITATION CONTRACTION COUPLING; GENE DELETION; GENOTYPE; HEART RATE; KINETICS; MICE; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; PHENOTYPE; RAS PROTEINS; SARCOMERES; UP-REGULATION; VENTRICULAR PRESSURE;

EID: 84902301881     PISSN: None     EISSN: 20479980     Source Type: Journal    
DOI: 10.1161/JAHA.113.000459     Document Type: Article

References (0)