Senescent cardiac fibroblast is critical for cardiac fibrosis after myocardial infarction.

PloS one

Volumn 8, Issue 9, 2013, Pages

  Zhu, Fuli ^a   Li, Yulin ^b   Zhang, Junmeng ^b   Piao, Chunmei ^b   Liu, Tingting ^b   Li, Hui Hua ^b   Du, Jie ^b  

^a CAPITAL MEDICAL UNIVERSITY   (China)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

COLLAGEN; PROTEIN P53; SMALL INTERFERING RNA;

ADENOVIRUS; ANIMAL; ARTICLE; CELL AGING; CELL CULTURE; CELL HYPOXIA; CELL MOTION; DISEASE MODEL; DRUG ANTAGONISM; FIBROBLAST; GENE EXPRESSION REGULATION; GENE VECTOR; GENETICS; HEART INFARCTION; HEART MUSCLE; HEART MUSCLE FIBROSIS; KNOCKOUT MOUSE; MALE; METABOLISM; MOUSE; PATHOLOGY; SIGNAL TRANSDUCTION;

ADENOVIRIDAE; ANIMALS; CELL AGING; CELL HYPOXIA; CELL MOVEMENT; CELLS, CULTURED; COLLAGEN; DISEASE MODELS, ANIMAL; ENDOMYOCARDIAL FIBROSIS; FIBROBLASTS; GENE EXPRESSION REGULATION; GENETIC VECTORS; MALE; MICE; MICE, KNOCKOUT; MYOCARDIAL INFARCTION; MYOCARDIUM; RNA, SMALL INTERFERING; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR PROTEIN P53;

EID: 84901733241     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0074535     Document Type: Article

References (0)