Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis.

Cell death & disease

Volumn 5, Issue , 2014, Pages

  Remijsen, Q ^a   Goossens, V ^a   Grootjans, S ^a   Van den Haute, C ^a   Vanlangenakker, N ^a   Dondelinger, Y ^a   Roelandt, R ^a   Bruggeman, I ^a   Goncalves, A ^a   Bertrand, M J ^a   Baekelandt, V ^a   Takahashi, N ^a   Berghe, T V ^a   Vandenabeele, P ^a  

^a GHENT UNIVERSITY   (Belgium)

Author keywords

[No Author keywords available]

Indexed keywords

DNM1L PROTEIN, MOUSE; DYNAMIN; MLKL PROTEIN, MOUSE; PGAM5 PROTEIN, MOUSE; PHOSPHATASE; PROTEIN KINASE; RECEPTOR INTERACTING PROTEIN SERINE THREONINE KINASE; RIPK1 PROTEIN, MOUSE; RIPK3 PROTEIN, MOUSE; TUMOR NECROSIS FACTOR;

ANIMAL; APOPTOSIS; ARTICLE; GENETICS; HUMAN; METABOLISM; MOUSE; NECROSIS;

ANIMALS; APOPTOSIS; DYNAMINS; HUMANS; MICE; NECROSIS; PHOSPHORIC MONOESTER HYDROLASES; PROTEIN KINASES; RECEPTOR-INTERACTING PROTEIN SERINE-THREONINE KINASES; TUMOR NECROSIS FACTORS;

EID: 84901257054     PISSN: None     EISSN: 20414889     Source Type: Journal    
DOI: 10.1038/cddis.2013.531     Document Type: Article

References (0)