The environment-pathogen-host axis in communicable and non-communicable diseases: Recent advances in experimental and clinical research;Die Achse Umwelt-Erreger-Wirt bei übertragbaren und nicht übertragbaren Krankheiten: Jüngste Fortschritte in der experimentellen und klinischen Forschung

JDDG - Journal of the German Society of Dermatology

Volumn 12, Issue 5, 2014, Pages 395-399

  Gilles, Stefanie ^a,b   Traidl Hoffmann, Claudia ^a,b  

^a UNIKA T   (Germany)

^b CK CARE   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

ALLERGEN;

ALLERGY; ASTHMA; ATOPIC DERMATITIS; CLINICAL RESEARCH; COMMUNICABLE DISEASE; ENVIRONMENTAL CHANGE; ENVIRONMENTAL FACTOR; GREENHOUSE EFFECT; HOSPITAL PATIENT; HOST PATHOGEN INTERACTION; HUMAN; IMMUNOSUPPRESSIVE TREATMENT; INTESTINE; MICROFLORA; MUCOCUTANEOUS CANDIDIASIS; NON COMMUNICABLE DISEASE; NONHUMAN; POLLEN; POLLUTION; REVIEW; SKIN; TRANSLATIONAL RESEARCH; URBANIZATION; AUTOIMMUNE DISEASES; CLIMATE CHANGE; DISEASE CARRIER; ENVIRONMENTAL EXPOSURE; ENVIRONMENTAL HEALTH; FORECASTING; GENETICALLY MODIFIED FOOD; HYPERSENSITIVITY; POPULATION DYNAMICS; RISK FACTOR; TRANSMISSION; TRENDS;

AUTOIMMUNE DISEASES; CLIMATE CHANGE; COMMUNICABLE DISEASES; DISEASE RESERVOIRS; ENVIRONMENTAL EXPOSURE; ENVIRONMENTAL MEDICINE; ENVIRONMENTAL POLLUTION; FOOD, GENETICALLY MODIFIED; FORECASTING; HUMANS; HYPERSENSITIVITY; POPULATION DYNAMICS; RISK FACTORS; TRANSLATIONAL MEDICAL RESEARCH; URBANIZATION;

EID: 84900793367     PISSN: 16100379     EISSN: 16100387     Source Type: Journal    
DOI: 10.1111/ddg.12345     Document Type: Review

References (21)

1. Ring, J, Akdis C, Behrendt H, et al. Davos declaration: allergy as a global problem. Allergy 2012; 67 (2): 141-3.
2. Buters JT, Kasche A, Weichenmeier I, et al. Year-to-year variation in release of Bet v 1 allergen from birch pollen: evidence for geographical differences between West and South Germany. Int Arch Allergy Immunol 2008; 145 (2): 122-30.
3. Braun-Fahrlander C, Riedler J, Herz U, et al. Environmental exposure to endotoxin and its relation to asthma in school-age children. N Engl J Med 2002; 347 (12): 869-77.
4. Eyerich K, Eyerich S, Hiller J, et al. Chronic mucocutaneous candidiasis, from bench to bedside. Eur J Dermatol 2010; 20 (3): 260-5.
5. Eyerich K, Rombold S, Foerster S, et al. Altered, but not diminished specific T cell response in chronic mucocutaneous candidiasis patients. Arch Dermatol Res 2007; 299 (10): 475-81.
6. Eyerich K, Rombold S, Foerster S, et al. Patients with chronic mucocutaneous candidiasis exhibit reduced production of Th17-associated cytokines IL-17 and IL-22. J Invest Dermatol 2008; 128 (11): 2640-5.
7. Liu L, Okada S, Kong XF, et al. Gain-of-function human STAT1 mutations impair IL-17 immunity and underlie chronic mucocutaneous candidiasis. J Exp Med 2011; 208 (8): 1635-48.
8. Gilles S, Behrendt H, Ring J, Traidl-Hoffmann C,. Pollen allergens do not come alone: pollen associated lipid mediators (PALMS) shift the human immune systems towards a T(H)2-dominated response. Allergy Asthma Clin Immunol 2009; 5 (1): 3.
9. Plotz SG, Traidl-Hoffmann C, Feussner I, et al. Chemotaxis and activation of human peripheral blood eosinophils induced by pollen-associated lipid mediators. J Allergy Clin Immunol 2004; 113 (6): 1152-60.
10. Traidl-Hoffmann C, Kasche A, Jakob T, et al. Lipid mediators from pollen act as chemoattractants and activators of polymorphonuclear granulocytes. J Allergy Clin Immunol 2002; 109 (5): 831-8.
11. Gilles S, Jacoby D, Blume C, et al. Pollen-derived low-molecular weight factors inhibit 6-sulfo LacNAc+ dendritic cells' capacity to induce T-helper type 1 responses. Clin Exp Allergy 2010; 40 (2): 269-78.
12. Gilles S, Mariani V, Bryce M, et al. Pollen-derived E1-phytoprostanes signal via PPAR-gamma and NF-kappaB-dependent mechanisms. J Immunol 2009; 182 (11): 6653-8.
13. Traidl-Hoffmann C, Mariani V, Hochrein H, et al. Pollen-associated phytoprostanes inhibit dendritic cell interleukin-12 production and augment T helper type 2 cell polarization. J Exp Med 2005; 201 (4): 627-36.
14. Mariani V, Gilles S, Jakob T, et al. Immunomodulatory mediators from pollen enhance the migratory capacity of dendritic cells and license them for Th2 attraction. J Immunol 2007; 178 (12): 7623-31.
15. Gutermuth J, Bewersdorff M, Traidl-Hoffmann C, et al. Immunomodulatory effects of aqueous birch pollen extracts and phytoprostanes on primary immune responses in vivo. J Allergy Clin Immunol 2007; 120 (2): 293-9.
16. Metz M, Gilles S, Geldmacher A, et al. Evidence for non-allergic mast cell activation in pollen-associated inflammation. J Invest Dermatol 2011; 131 (4): 987-90.
17. Gilles S, Fekete A, Zhang X, et al. Pollen metabolome analysis reveals adenosine as a major regulator of dendritic cell-primed T(H) cell responses. J Allergy Clin Immunol 2011; 127 (2): 454-461 e1-9.
18. Alessandrini F, Schulz H, Takenaka S, et al. Effects of ultrafine carbon particle inhalation on allergic inflammation of the lung. J Allergy Clin Immunol 2006; 117 (4): 824-30.
19. Alessandrini F, Weichenmeier I, van Miert E, et al. Effects of ultrafine particles-induced oxidative stress on Clara cells in allergic lung inflammation. Part Fibre Toxicol 2010; 7: 11.
20. D'Amato G, Liccardi G, D'Amato M, Cazzola M,. Outdoor air pollution, climatic changes and allergic bronchial asthma. Eur Respir J 2002; 20 (3): 763-76.
21. Beck I, Jochner S, Gilles S, et al. High environmental ozone levels lead to enhanced allergenicity of birch pollen. PLoS One 2013; 8 (11): e80147.