Elimination of NADPH oxidase activity promotes reductive stress and sensitizes the heart to ischemic injury.

Journal of the American Heart Association

Volumn 3, Issue 1, 2014, Pages

  Yu, Qiujun ^a   Lee, Chi Fung ^b   Wang, Wang ^b   Karamanlidis, Georgios ^b   Kuroda, Junya ^b   Matsushima, Shouji ^b   Sadoshima, Junichi ^b   Tian, Rong ^b  

^a UNIVERSITY OF WASHINGTON   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

GLUTATHIONE; NICOTINAMIDE ADENINE DINUCLEOTIDE; NOX4 PROTEIN, MOUSE; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;

ANIMAL; ARTICLE; CARDIAC ISCHEMIC INJURY; DISEASE MODEL; ENERGY METABOLISM; ENZYMOLOGY; GENETICS; HEART CONTRACTION; HEART INFARCTION; HEART MITOCHONDRION; HEART MUSCLE; KNOCKOUT MOUSE; METABOLISM; MOUSE; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PATHOLOGY; PATHOPHYSIOLOGY; REDUCTIVE STRESS; REPERFUSION INJURY; TIME;

CARDIAC ISCHEMIC INJURY; NADPH OXIDASE; OXIDATIVE STRESS; REDUCTIVE STRESS;

ANIMALS; DISEASE MODELS, ANIMAL; ENERGY METABOLISM; GLUTATHIONE; MICE; MICE, KNOCKOUT; MITOCHONDRIA, HEART; MYOCARDIAL CONTRACTION; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; NAD; NADPH OXIDASE; OXIDATION-REDUCTION; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES; TIME FACTORS;

EID: 84898631692     PISSN: None     EISSN: 20479980     Source Type: Journal    
DOI: 10.1161/JAHA.113.000555     Document Type: Article

References (0)