-
1
-
-
33748132658
-
A prospective study of the prevalence of primary aldosteronism in 1,125 hypertensive patients
-
Rossi GP, Bernini G, Caliumi C, et al A prospective study of the prevalence of primary aldosteronism in 1,125 hypertensive patients. J Am Coll Cardiol. 2006;48(11):2293-2300.
-
(2006)
J Am Coll Cardiol.
, vol.48
, Issue.11
, pp. 2293-2300
-
-
Rossi, G.P.1
Bernini, G.2
Caliumi, C.3
-
2
-
-
18844464024
-
Control of aldosterone secretion: A model for convergence in cellular signaling pathways
-
Spat A, Hunyady L. Control of aldosterone secretion: a model for convergence in cellular signaling pathways. Physiol Rev . 2004; 84(2):489-539.
-
(2004)
Physiol Rev.
, vol.84
, Issue.2
, pp. 489-539
-
-
Spat, A.1
Hunyady, L.2
-
3
-
-
0029809279
-
Distinct functions of T-and L-type calcium channels during activation of bovine adrenal glomerulosa cells
-
Rossier MF, Burnay MM, Vallotton MB, Capponi AM. Distinct functions of T-and L-type calcium channels during activation of bovine adrenal glomerulosa cells. Endocrinology . 1996;137(11): 4817-4826.
-
(1996)
Endocrinology.
, vol.137
, Issue.11
, pp. 4817-4826
-
-
Rossier, M.F.1
Burnay, M.M.2
Vallotton, M.B.3
Capponi, A.M.4
-
4
-
-
0035020221
-
1H T-type Ca2 channel is the predominant subtype expressed in bovine and rat zona glomerulosa
-
Schrier AD, Wang H, Talley EM, Perez-Reyes E, Barrett PQ. 1H T-type Ca2- channel is the predominant subtype expressed in bovine and rat zona glomerulosa. Am J Physiol Cell Physiol . 2001; 280(2):C265-C272.
-
(2001)
Am J Physiol Cell Physiol
, vol.280
, Issue.2
-
-
Schrier, A.D.1
Wang, H.2
Talley, E.M.3
Perez-Reyes, E.4
Barrett, P.Q.5
-
5
-
-
79951506090
-
K channel mutations in adrenal aldosterone-producing adenomas and hereditary hypertension
-
Choi M, Scholl UI, Yue P, et al. K channel mutations in adrenal aldosterone-producing adenomas and hereditary hypertension. Science . 2011;331(6018):768-772.
-
(2011)
Science
, vol.331
, Issue.6018
, pp. 768-772
-
-
Choi, M.1
Scholl, U.I.2
Yue, P.3
-
6
-
-
84863393293
-
Somatic mutations affecting the selectivity filter of KCNJ5 are frequent in 2 large unselected collections of adrenal aldosteronomas
-
Azizan EA, Murthy M, Stowasser M, et al. Somatic mutations affecting the selectivity filter of KCNJ5 are frequent in 2 large unselected collections of adrenal aldosteronomas. Hypertension . 2012; 59(3):587-591.
-
(2012)
Hypertension.
, vol.59
, Issue.3
, pp. 587-591
-
-
Azizan, E.A.1
Murthy, M.2
Stowasser, M.3
-
7
-
-
84857623168
-
Prevalence, clinical, and molecular correlates ofKCNJ5mutations in primary aldosteronism
-
Boulkroun S, Beuschlein F, Rossi GP, et al. Prevalence, clinical, and molecular correlates ofKCNJ5mutations in primary aldosteronism. Hypertension . 2012;59(3):592-598.
-
(2012)
Hypertension.
, vol.59
, Issue.3
, pp. 592-598
-
-
Boulkroun, S.1
Beuschlein, F.2
Rossi, G.P.3
-
8
-
-
84864612057
-
Comprehensive re-sequencing of adrenal aldosterone producing lesions reveal three somatic mutations near the KCNJ5 potassium channel selectivity filter
-
Akerstrom T, Crona J, Delgado Verdugo A, et al. Comprehensive re-sequencing of adrenal aldosterone producing lesions reveal three somatic mutations near the KCNJ5 potassium channel selectivity filter. PLoS One . 2012;7(7):e41926.
-
(2012)
PLoS One
, vol.7
, Issue.7
-
-
Akerstrom, T.1
Crona, J.2
Delgado Verdugo, A.3
-
9
-
-
84859378136
-
Expression and mutations of KCNJ5 mRNA in Japanese patients with aldosterone-producing adenomas
-
Taguchi R, Yamada M, Nakajima Y, et al. Expression and mutations of KCNJ5 mRNA in Japanese patients with aldosterone-producing adenomas. J Clin Endocrinol Metab . 2012;97(4):1311-1319.
-
(2012)
J Clin Endocrinol Metab.
, vol.97
, Issue.4
, pp. 1311-1319
-
-
Taguchi, R.1
Yamada, M.2
Nakajima, Y.3
-
10
-
-
84862640301
-
KCNJ5 mutations in the national institutes of health cohort of patients with primary hyperaldosteronism: An infrequent genetic cause of Conns syndrome
-
Xekouki P, Hatch MM, Lin L, et al. KCNJ5 mutations in the national institutes of health cohort of patients with primary hyperaldosteronism: an infrequent genetic cause of Conns syndrome. Endocr Relat Cancer . 2012;19(3):255-260.
-
(2012)
Endocr Relat Cancer.
, vol.19
, Issue.3
, pp. 255-260
-
-
Xekouki, P.1
Hatch, M.M.2
Lin, L.3
-
11
-
-
84867917709
-
KCNJ5 mutations in aldosterone-and cortisol-co-secreting adrenal adenomas
-
Yamada M, Nakajima Y, Taguchi R, et al. KCNJ5 mutations in aldosterone-and cortisol-co-secreting adrenal adenomas. Endocr J . 2012;59(8):735-741.
-
(2012)
Endocr J.
, vol.59
, Issue.8
, pp. 735-741
-
-
Yamada, M.1
Nakajima, Y.2
Taguchi, R.3
-
12
-
-
54349104413
-
Emerging roles for two-pore-domain potassium channels and their potential therapeutic impact
-
Bayliss DA, Barrett PQ. Emerging roles for two-pore-domain potassium channels and their potential therapeutic impact. Trends Pharmacol Sci . 2008;29(11):566-575.
-
(2008)
Trends Pharmacol Sci.
, vol.29
, Issue.11
, pp. 566-575
-
-
Bayliss, D.A.1
Barrett, P.Q.2
-
13
-
-
41149160175
-
TASK channel deletion in mice causes primary hyperaldosteronism
-
Davies LA, Hu C, Guagliardo NA, et al. TASK channel deletion in mice causes primary hyperaldosteronism. Proc Natl Acad Sci USA . 2008;105(6):2203- 2208.
-
(2008)
Proc Natl Acad Sci USA.
, vol.105
, Issue.6
, pp. 2203-2208
-
-
Davies, L.A.1
Hu, C.2
Guagliardo, N.A.3
-
14
-
-
38049015880
-
Invalidation of TASK1 potassium channels disrupts adrenal gland zonation and mineralocorticoid homeostasis
-
Heitzmann D, Derand R, Jungbauer S, et al. Invalidation of TASK1 potassium channels disrupts adrenal gland zonation and mineralocorticoid homeostasis. EMBO J . 2008;27(1):179-187.
-
(2008)
EMBO J.
, vol.27
, Issue.1
, pp. 179-187
-
-
Heitzmann, D.1
Derand, R.2
Jungbauer, S.3
-
15
-
-
84860223991
-
TASK-3 channel deletion in mice recapitulates low-renin essential hypertension
-
Guagliardo NA, Yao J,HuC, et al. TASK-3 channel deletion in mice recapitulates low-renin essential hypertension. Hypertension . 2012; 59(5):999-1005.
-
(2012)
Hypertension.
, vol.59
, Issue.5
, pp. 999-1005
-
-
Guagliardo, N.A.1
Yao, J.2
Hu, C.3
-
16
-
-
84880653433
-
Severe hyperaldosteronism in neonatal Task3 potassium channel knockout mice is associated with activation of the intra-adrenal renin-angiotensin system
-
Bandulik S, Tauber P, Penton D, et al. Severe hyperaldosteronism in neonatal Task3 potassium channel knockout mice is associated with activation of the intra-adrenal renin-angiotensin system. Endocrinology. 2013;154(8):2712- 2722.
-
(2013)
Endocrinology.
, vol.154
, Issue.8
, pp. 2712-2722
-
-
Bandulik, S.1
Tauber, P.2
Penton, D.3
-
17
-
-
84866754221
-
Task3 potassium channel gene invalidation causes low renin and salt-sensitive arterial hypertension
-
Penton D, Bandulik S, Schweda F, et al. Task3 potassium channel gene invalidation causes low renin and salt-sensitive arterial hypertension. Endocrinology . 2012;153(10):4740-4748.
-
(2012)
Endocrinology.
, vol.153
, Issue.10
, pp. 4740-4748
-
-
Penton, D.1
Bandulik, S.2
Schweda, F.3
-
18
-
-
77953740440
-
The role of TASK1 in aldosterone production and its expression in normal adrenal and aldosterone-producing adenomas
-
Nogueira EF, Gerry D, Mantero F, Mariniello B, Rainey WE. The role of TASK1 in aldosterone production and its expression in normal adrenal and aldosterone-producing adenomas. Clin Endocrinol (Oxf) . 2010;73(1):22-29.
-
(2010)
Clin Endocrinol (Oxf).
, vol.73
, Issue.1
, pp. 22-29
-
-
Nogueira, E.F.1
Gerry, D.2
Mantero, F.3
Mariniello, B.4
Rainey, W.E.5
-
19
-
-
36448930836
-
Heterogeneity of aldosterone-producing adenomas revealed by a whole transcriptome analysis
-
Lenzini L, Seccia TM, Aldighieri E, et al. Heterogeneity of aldosterone-producing adenomas revealed by a whole transcriptome analysis. Hypertension . 2007;50(6):1106-1113.
-
(2007)
Hypertension.
, vol.50
, Issue.6
, pp. 1106-1113
-
-
Lenzini, L.1
Seccia, T.M.2
Aldighieri, E.3
-
20
-
-
84870748191
-
Somatic mutations in the KCNJ5 gene raise the lateralization index: Implications for the diagnosis of primary aldosteronism by adrenal vein sampling
-
Seccia TM, Mantero F, Letizia C, et al. Somatic mutations in the KCNJ5 gene raise the lateralization index: implications for the diagnosis of primary aldosteronism by adrenal vein sampling. J Clin Endocrinol Metab . 2012;97(12):E2307-E2313.
-
(2012)
J Clin Endocrinol Metab.
, vol.97
, Issue.12
-
-
Seccia, T.M.1
Mantero, F.2
Letizia, C.3
-
21
-
-
0036188942
-
TASK-3 dominates the background potassium conductance in rat adrenal glomerulosa cells
-
Czirjak G, Enyedi P. TASK-3 dominates the background potassium conductance in rat adrenal glomerulosa cells. Mol Endocrinol . 2002;16(3):621-629.
-
(2002)
Mol Endocrinol.
, vol.16
, Issue.3
, pp. 621-629
-
-
Czirjak, G.1
Enyedi, P.2
-
22
-
-
0034465794
-
TASK (TWIKrelated acid-sensitive K- channel) is expressed in glomerulosa cells of rat adrenal cortex and inhibited by angiotensin II
-
Czirjak G, Fischer T, Spat A, Lesage F, Enyedi P. TASK (TWIKrelated acid-sensitive K- channel) is expressed in glomerulosa cells of rat adrenal cortex and inhibited by angiotensin II. Mol Endocrinol . 2000;14(6):863-874.
-
(2000)
Mol Endocrinol.
, vol.14
, Issue.6
, pp. 863-874
-
-
Czirjak, G.1
Fischer, T.2
Spat, A.3
Lesage, F.4
Enyedi, P.5
-
23
-
-
0026580019
-
A chimaeric 11-hydroxylase/ aldosterone synthase gene causes glucocorticoid-remediable aldosteronism and human hypertension
-
Lifton RP, Dluhy RG, Powers M, et al. A chimaeric 11-hydroxylase/ aldosterone synthase gene causes glucocorticoid-remediable aldosteronism and human hypertension. Nature . 1992;355(6357): 262-265.
-
(1992)
Nature.
, vol.355
, Issue.6357
, pp. 262-265
-
-
Lifton, R.P.1
Dluhy, R.G.2
Powers, M.3
-
24
-
-
84860776956
-
Microarray, qPCR, and KCNJ5 sequencing of aldosterone-producing adenomas reveal differences in genotype and phenotype between zona glomerulosa-and zona fasciculata-like tumors
-
Azizan EA, Lam BY, Newhouse SJ, et al. Microarray, qPCR, and KCNJ5 sequencing of aldosterone-producing adenomas reveal differences in genotype and phenotype between zona glomerulosa-and zona fasciculata-like tumors. J Clin Endocrinol Metab . 2012;97(5): E819-E829.
-
(2012)
J Clin Endocrinol Metab.
, vol.97
, Issue.5
-
-
Azizan, E.A.1
Lam, B.Y.2
Newhouse, S.J.3
-
25
-
-
0032553319
-
Cloning and expression of a novel pH-sensitive two pore domain K- channel from human kidney
-
Reyes R, Duprat F, Lesage F, et al. Cloning and expression of a novel pH-sensitive two pore domain K- channel from human kidney. J Biol Chem . 1998;273(47):30863-30869.
-
(1998)
J Biol Chem.
, vol.273
, Issue.47
, pp. 30863-30869
-
-
Reyes, R.1
Duprat, F.2
Lesage, F.3
-
26
-
-
57349180044
-
Both TASK-3 and TREK-1 twopore loop K channels are expressed in H295R cells and modulate their membrane potential and aldosterone secretion
-
Brenner T, OShaughnessy KM. Both TASK-3 and TREK-1 twopore loop K channels are expressed in H295R cells and modulate their membrane potential and aldosterone secretion. Am J Physiol Endocrinol Metab . 2008;295(6):E1480-E1486.
-
(2008)
Am J Physiol Endocrinol Metab.
, vol.295
, Issue.6
-
-
Brenner, T.1
Oshaughnessy, K.M.2
-
27
-
-
2542555065
-
Proximal renal tubular acidosis in TASK2K-channel-deficient mice reveals a mechanism for stabilizing bicarbonate transport
-
Warth R, Barriere H, Meneton P, et al. Proximal renal tubular acidosis in TASK2K-channel-deficient mice reveals a mechanism for stabilizing bicarbonate transport. Proc Natl Acad Sci USA . 2004; 101(21):8215-8220.
-
(2004)
Proc Natl Acad Sci USA.
, vol.101
, Issue.21
, pp. 8215-8220
-
-
Warth, R.1
Barriere, H.2
Meneton, P.3
-
28
-
-
84883291331
-
MicroRNA-24 is a novel regulator of aldosterone and cortisol production in the human adrenal cortex
-
Robertson S, Mackenzie SM, Alvarez-Madrazo S, et al. MicroRNA-24 is a novel regulator of aldosterone and cortisol production in the human adrenal cortex. Hypertension . 2013;62(3):572-578.
-
(2013)
Hypertension.
, vol.62
, Issue.3
, pp. 572-578
-
-
Robertson, S.1
Mackenzie, S.M.2
Alvarez-Madrazo, S.3
-
29
-
-
84876088023
-
KCNJ5 mudoi tations in aldosterone producing adenoma and relationship with adrenal cortex remodeling
-
Boulkroun S, Golib Dzib JF, Samson-Couterie B, et al. KCNJ5 mudoi tations in aldosterone producing adenoma and relationship with adrenal cortex remodeling. Mol Cell Endocrinol . 2013;371(1-2): 221-227.
-
(2013)
Mol Cell Endocrinol
, vol.371
, Issue.1-2
, pp. 221-227
-
-
Boulkroun, S.1
Golib Dzib, J.F.2
Samson-Couterie, B.3
-
30
-
-
84864592834
-
A novel point mutation in the KCNJ5 gene causing primary hyperaldosteronism and earlyonset autosomal dominant hypertension
-
Charmandari E, Sertedaki A, Kino T, et al. A novel point mutation in the KCNJ5 gene causing primary hyperaldosteronism and earlyonset autosomal dominant hypertension. J Clin Endocrinol Metab . 2012;97(8):E1532-9.
-
(2012)
J Clin Endocrinol Metab.
, vol.97
, Issue.8
-
-
Charmandari, E.1
Sertedaki, A.2
Kino, T.3
-
31
-
-
84873129212
-
Genetic variations in the KCNJ5 gene in primary aldosteronism patients from Xingjian, china
-
Li NF, Li HJ, Zhang DL, et al. Genetic variations in the KCNJ5 gene in primary aldosteronism patients from Xingjian, china. PLoS One . 2013;8(1):e54051.
-
(2013)
PLoS One
, vol.8
, Issue.1
-
-
Li, N.F.1
Li, H.J.2
Zhang, D.L.3
-
32
-
-
84864817326
-
Effect of KCNJ5 mutations on gene expression in aldosterone-producing adenomas and adrenocortical cells
-
Monticone S, Hattangady NG, Nishimoto K, et al. Effect of KCNJ5 mutations on gene expression in aldosterone-producing adenomas and adrenocortical cells. J Clin Endocrinol Metab . 2012;97(8): E1567-E1572.
-
(2012)
J Clin Endocrinol Metab.
, vol.97
, Issue.8
-
-
Monticone, S.1
Hattangady, N.G.2
Nishimoto, K.3
-
33
-
-
84856300896
-
KCNJ5 mutations in European families with nonglucocorticoid remediable familial hyperaldosteronism
-
Mulatero P, Tauber P, Zennaro MC, et al. KCNJ5 mutations in European families with nonglucocorticoid remediable familial hyperaldosteronism. Hypertension . 2012;59(2):235-240.
-
(2012)
Hypertension.
, vol.59
, Issue.2
, pp. 235-240
-
-
Mulatero, P.1
Tauber, P.2
Zennaro, M.C.3
-
34
-
-
84865495951
-
Characterization of a novel somatic KCNJ5 mutation delI157 in an aldosterone-producing adenoma
-
Murthy M, Azizan EA, Brown MJ, OShaughnessy KM. Characterization of a novel somatic KCNJ5 mutation delI157 in an aldosterone-producing adenoma. J Hypertens . 2012;30(9):1827-1833.
-
(2012)
J Hypertens.
, vol.30
, Issue.9
, pp. 1827-1833
-
-
Murthy, M.1
Azizan, E.A.2
Brown, M.J.3
Oshaughnessy, K.M.4
-
35
-
-
84859410866
-
Potassium channel mutant KCNJ5 T158A expression in HAC-15 cells increases aldosterone synthesis
-
Oki K, Plonczynski MW, Luis Lam M, Gomez-Sanchez EP, Gomez-Sanchez CE. Potassium channel mutant KCNJ5 T158A expression in HAC-15 cells increases aldosterone synthesis. Endocrinology . 2012;153(4):1774-1782.
-
(2012)
Endocrinology.
, vol.153
, Issue.4
, pp. 1774-1782
-
-
Oki, K.1
Plonczynski, M.W.2
Luis Lam, M.3
Gomez-Sanchez, E.P.4
Gomez-Sanchez, C.E.5
-
36
-
-
84865427053
-
The potassium channel, Kir3.4 participates in angiotensin II-stimulated aldosterone production by a human adrenocortical cell line
-
Oki K, Plonczynski MW, Lam ML, Gomez-Sanchez EP, Gomez-Sanchez CE. The potassium channel, Kir3.4 participates in angiotensin II-stimulated aldosterone production by a human adrenocortical cell line. Endocrinology . 2012;153(9):4328-4335.
-
(2012)
Endocrinology.
, vol.153
, Issue.9
, pp. 4328-4335
-
-
Oki, K.1
Plonczynski, M.W.2
Lam, M.L.3
Gomez-Sanchez, E.P.4
Gomez-Sanchez, C.E.5
-
37
-
-
84873567626
-
New insights into aldosterone-producing adenomas and hereditary aldosteronism: Mutations in the K-channel KCNJ5
-
Scholl UI, Lifton RP. New insights into aldosterone-producing adenomas and hereditary aldosteronism: mutations in the K-channel KCNJ5. Curr Opin Nephrol Hypertens . 2013;22(2):141-147.
-
(2013)
Curr Opin Nephrol Hypertens.
, vol.22
, Issue.2
, pp. 141-147
-
-
Scholl, U.I.1
Lifton, R.P.2
-
38
-
-
84863115868
-
Hypertension with or without adrenal hyperplasia due to different inherited mutations in the potassium channel KCNJ5
-
Scholl UI, Nelson-Williams C, Yue P, et al. Hypertension with or without adrenal hyperplasia due to different inherited mutations in the potassium channel KCNJ5. Proc Natl Acad Sci USA . 2012; 109(7):2533-2538.
-
(2012)
Proc Natl Acad Sci USA.
, vol.109
, Issue.7
, pp. 2533-2538
-
-
Scholl, U.I.1
Nelson-Williams, C.2
Yue, P.3
|