Is subchondral bone the crucial point for the pathogenesis and the treatment of osteoarthritis?

Eklem Hastaliklari ve Cerrahisi

Volumn 25, Issue 1, 2014, Pages 1-

  Sahap Atik O ^a  

^a GAZI UNIVERSITY   (Turkey)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 84897845504     PISSN: 13058282     EISSN: None     Source Type: Journal    
DOI: 10.5606/ehc.2014.01     Document Type: Editorial

References (7)

1. Martel-Pelletier J, Pelletier JP. Is osteoarthritis a disease involving only cartilage or other articular tissues?. Eklem Hastalik Cerrahisi 2010;21:2-14.
2. Sahap Atik O. Leukotriene B4 and prostaglandin E2-like activity in synovial fluid in osteoarthritis. Prostaglandins Leukot Essent Fatty Acids 1990;39:253-4.
3. Raynauld JP, Martel-Pelletier J, Berthiaume MJ, Abram F, Choquette D, Haraoui B, et al. Correlation between bone lesion changes and cartilage volume loss in patients with osteoarthritis of the knee as assessed by quantitative magnetic resonance imaging over a 24-month period. Ann Rheum Dis 2008;67:683-8.
4. Atik OŞ, Tokgöz N. Do periarticular dense bone islands cause cartilage destruction?. Eklem Hastalik Cerrahisi 2013;24:39-40.
5. Suri S, Walsh DA. Osteochondral alterations in osteoarthritis. Bone 2012;51:204-11.
6. Bijlsma JW, Berenbaum F, Lafeber FP. Osteoarthritis: an update with relevance for clinical practice. Lancet 2011;377:2115-26.
7. Castañeda S, Roman-Blas JA, Largo R, Herrero-Beaumont G. Subchondral bone as a key target for osteoarthritis treatment. Biochem Pharmacol 2012;83:315-23.