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Volumn 45, Issue 4, 2014, Pages 1139-1147
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Chelating intracellularly accumulated zinc decreased ischemic brain injury through reducing neuronal apoptotic death
a,b,c,d e a,b a,b,c,d a,b,c a,c,d a,b,c,d a,b a,b,c,d e |
Author keywords
[No Author keywords available]
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Indexed keywords
N,N,N',N' TETRAKIS(2 PYRIDYLMETHYL)ETHYLENEDIAMINE;
NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE 1;
ZINC;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
BRAIN DAMAGE;
BRAIN INFARCTION SIZE;
BRAIN INJURY;
BRAIN ISCHEMIA;
CELL DEATH;
CHELATION THERAPY;
CLINICAL ASSESSMENT;
CONTROLLED STUDY;
ENZYME ACTIVITY;
MALE;
MIDDLE CEREBRAL ARTERY;
MIDDLE CEREBRAL ARTERY OCCLUSION;
MOTOR PERFORMANCE;
NERVE CELL NECROSIS;
NEUROPROTECTION;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
WESTERN BLOTTING;
BRAIN ISCHEMIA;
INFARCTION, MIDDLE CEREBRAL ARTERY;
POLY(ADP-RIBOSE) POLYMERASES;
ZINC;
ANIMALS;
APOPTOSIS;
BRAIN ISCHEMIA;
CHELATING AGENTS;
CYTOSOL;
ETHYLENEDIAMINES;
INFARCTION, MIDDLE CEREBRAL ARTERY;
ISCHEMIC ATTACK, TRANSIENT;
MALE;
MOTOR ACTIVITY;
NEURONS;
NEUROPROTECTIVE AGENTS;
POLY(ADP-RIBOSE) POLYMERASES;
RATS;
RATS, SPRAGUE-DAWLEY;
ZINC;
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EID: 84897425078
PISSN: 00392499
EISSN: 15244628
Source Type: Journal
DOI: 10.1161/STROKEAHA.113.004296 Document Type: Article |
Times cited : (58)
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References (0)
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