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Volumn 114, Issue 2, 2014, Pages 283-294
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Increased β-adrenergic inotropy in ventricular myocardium from Trpm4-/- mice.
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Author keywords
[No Author keywords available]
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Indexed keywords
BETA ADRENERGIC RECEPTOR;
BETA ADRENERGIC RECEPTOR STIMULATING AGENT;
CALCIUM;
CALCIUM CHANNEL BLOCKING AGENT;
CALCIUM CHANNEL L TYPE;
CARDIOTONIC AGENT;
ION CHANNEL;
ISOPRENALINE;
TRANSIENT RECEPTOR POTENTIAL CHANNEL M;
TRPM4 PROTEIN, MOUSE;
129 MOUSE;
ACTION POTENTIAL;
ANIMAL;
ARTICLE;
CALCIUM SIGNALING;
DOSE RESPONSE;
DRUG EFFECT;
EXCITATION CONTRACTION COUPLING;
GENE EXPRESSION REGULATION;
GENETICS;
HEART CONTRACTION;
HEART MUSCLE;
HEART VENTRICLE;
KINETICS;
KNOCKOUT MOUSE;
METABOLISM;
MOUSE;
ACTION POTENTIALS;
CALCIUM SIGNALING;
EXCITATION CONTRACTION COUPLING;
ION CHANNELS;
MICE, KNOCKOUT;
TRPM4 PROTEIN, MOUSE;
ACTION POTENTIALS;
ADRENERGIC BETA-AGONISTS;
ANIMALS;
CALCIUM;
CALCIUM CHANNEL BLOCKERS;
CALCIUM CHANNELS, L-TYPE;
CARDIOTONIC AGENTS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
EXCITATION CONTRACTION COUPLING;
GENE EXPRESSION REGULATION;
HEART VENTRICLES;
ISOPROTERENOL;
KINETICS;
MICE;
MICE, 129 STRAIN;
MICE, KNOCKOUT;
MYOCARDIAL CONTRACTION;
MYOCARDIUM;
RECEPTORS, ADRENERGIC, BETA;
TRPM CATION CHANNELS;
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EID: 84897108860
PISSN: None
EISSN: 15244571
Source Type: None
DOI: 10.1161/CIRCRESAHA.114.302835 Document Type: Article |
Times cited : (71)
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References (0)
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