Proliferation of parenchymal microglia is the main source of microgliosis after ischaemic stroke.

Brain : a journal of neurology

Volumn 136, Issue Pt 12, 2013, Pages 3578-3588

  Li, Ting ^a   Pang, Shengru ^b   Yu, Yongping ^b   Wu, Xiaoqian ^b   Guo, Jing ^b   Zhang, Shengxiang ^b  

^a LANZHOU UNIVERSITY   (China)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ACTIN BINDING PROTEIN; AIF1 PROTEIN, MOUSE; CALCIUM BINDING PROTEIN; CASP3 PROTEIN, MOUSE; CASPASE 3; CCR2 PROTEIN, MOUSE; CHEMOKINE RECEPTOR; CHEMOKINE RECEPTOR CCR2; CX3CR1 PROTEIN, MOUSE; GREEN FLUORESCENT PROTEIN;

ANIMAL; BRAIN; C57BL MOUSE; CELL PROLIFERATION; CEREBROVASCULAR ACCIDENT; CONNECTIVE TISSUE; DISEASE MODEL; GENE EXPRESSION REGULATION; GENETICS; GLIOSIS; METABOLISM; MICROGLIA; MOUSE; PATHOLOGY; PHOTOSTIMULATION; PHYSIOLOGY; TIME; TRANSGENIC MOUSE;

ANIMALS; BRAIN; CALCIUM-BINDING PROTEINS; CASPASE 3; CELL PROLIFERATION; CONNECTIVE TISSUE; DISEASE MODELS, ANIMAL; GENE EXPRESSION REGULATION; GLIOSIS; GREEN FLUORESCENT PROTEINS; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MICROFILAMENT PROTEINS; MICROGLIA; PHOTIC STIMULATION; RECEPTORS, CCR2; RECEPTORS, CHEMOKINE; STROKE; TIME FACTORS;

EID: 84893515578     PISSN: None     EISSN: 14602156     Source Type: Journal    
DOI: 10.1093/brain/awt287     Document Type: Article

References (0)