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Volumn 170, Issue 1, 2014, Pages 220-222

Intermittent BRAF-inhibitor therapy is a feasible option: Report of a patient with metastatic melanoma

Author keywords

[No Author keywords available]

Indexed keywords

B RAF KINASE; B RAF KINASE INHIBITOR; DABRAFENIB; DACARBAZINE; TRAMETINIB; VEMURAFENIB; ANTINEOPLASTIC AGENT; INDOLE DERIVATIVE; SULFONAMIDE;

EID: 84892399771     PISSN: 00070963     EISSN: 13652133     Source Type: Journal    
DOI: 10.1111/bjd.12615     Document Type: Letter
Times cited : (21)

References (10)
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  • 2
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  • 3
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    • Resistance to BRAF inhibitors: Unraveling mechanisms and future treatment options
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  • 4
    • 79956039274 scopus 로고    scopus 로고
    • Receptor tyrosine kinases and their activation in melanoma
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  • 5
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    • Nazarian, R.1    Shi, H.2    Wang, Q.3
  • 6
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    • Targeting BRAF in advanced melanoma: A first step towards manageable disease
    • Vultur A, Villanueva J, Herlyn M, et al,. Targeting BRAF in advanced melanoma: a first step towards manageable disease. Clin Cancer Res 2011; 17: 1658-63.
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  • 7
    • 78650008177 scopus 로고    scopus 로고
    • Acquired resistance to BRAF inhibitors mediated by a RAF kinase switch in melanoma can be overcome by cotargeting MEK and IGF-1R/PI3K
    • Villanueva J, Vultur A, Lee JT, et al,. Acquired resistance to BRAF inhibitors mediated by a RAF kinase switch in melanoma can be overcome by cotargeting MEK and IGF-1R/PI3K. Cancer Cell 2010; 18: 683-95.
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.