Nonmelanoma skin cancer is associated with reduced alzheimer disease risk

Neurology

Volumn 81, Issue 23, 2013, Pages 2056-

  Thinnes, Friedrich P ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

ABC TRANSPORTER; AMYLOID A PROTEIN; AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; VOLTAGE DEPENDENT ANION CHANNEL;

ALZHEIMER DISEASE; BLOOD BRAIN BARRIER; CANCER SURVIVOR; DISEASE ASSOCIATION; HUMAN; MALIGNANT TRANSFORMATION; NEUROPATHOLOGY; NON MELANOMA SKIN CANCER; NOTE; PRIORITY JOURNAL; RISK REDUCTION;

ALZHEIMER DISEASE; HUMANS; RISK FACTORS; SKIN NEOPLASMS;

EID: 84892389368     PISSN: 00283878     EISSN: 1526632X     Source Type: Journal    
DOI: 10.1212/01.wnl.0000436924.94798.59     Document Type: Note

References (5)

1. White RS, Lipton RB, Hall CB, Steinerman JR. Nonmelanoma skin cancer is associated with reduced Alzheimer disease risk. Neurology 2013;80:1966-1972
2. Thinnes FP. Amyloid A-beta cut from APP by beta secretase BACE1 and gamma secretase induces apoptosis via opening type-1 porin/VDAC in cell membranes of hypometabolic cells-A basic model for the induction of apoptosis.Mol Genet Metab 2010;101:301-303
3. Thinnes FP. Apoptogenic interactions of plasmalemmal type-1 VDAC and A-beta peptides via GxxxG motifs induce Alzheimer's disease: a basic model of apoptosis. Wien Med Wochenschr 2011;161:274-276
4. Mattson MP, Begley JG, Mark RJ, Furukawa K. Abeta 25-35 induces rapid lysis of red blood cells: contrast with Abeta1-42 and examination of underlying mechanisms. Brain Res 1997;771:147-153
5. Thinnes FP. New findings concerning vertebrate porin II on the relevance of glycine motifs of type-1 VDAC. Mol Genet Metab 2013;108:212-224