Traumatic stress reactivity promotes excessive alcohol drinking and alters the balance of prefrontal cortex-amygdala activity.

Translational psychiatry

Volumn 3, Issue , 2013, Pages

  Edwards, S ^a   Baynes, B B ^b   Carmichael, C Y ^b   Zamora Martinez, E R ^b   Barrus, M ^b   Koob, G F ^b   Gilpin, N W ^b  

^a SCRIPPS RESEARCH INSTITUTE   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE;

ALCOHOLISM; AMYGDALOID NUCLEUS; ANIMAL; ARTICLE; AVOIDANCE BEHAVIOR; COMPULSION; CONDITIONED REFLEX; DISEASE MODEL; DRINKING BEHAVIOR; INSTRUMENTAL CONDITIONING; MALE; MENTAL STRESS; METABOLISM; PHOSPHORYLATION; POSTTRAUMATIC STRESS DISORDER; PREFRONTAL CORTEX; PSYCHOLOGICAL ASPECT; RAT; SIGNAL TRANSDUCTION; WISTAR RAT;

ALCOHOL DRINKING; ALCOHOLISM; AMYGDALA; ANIMALS; AVOIDANCE LEARNING; COMPULSIVE BEHAVIOR; CONDITIONING, CLASSICAL; CONDITIONING, OPERANT; DISEASE MODELS, ANIMAL; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; MALE; MAP KINASE SIGNALING SYSTEM; PHOSPHORYLATION; PREFRONTAL CORTEX; RATS; RATS, WISTAR; STRESS DISORDERS, POST-TRAUMATIC; STRESS, PSYCHOLOGICAL;

EID: 84889864708     PISSN: None     EISSN: 21583188     Source Type: Journal    
DOI: 10.1038/tp.2013.70     Document Type: Article

References (0)