How T cells trigger the dissociation of the endothelial receptor phosphatase VE-PTP from VE-cadherin
Vockel M, Vestweber D. How T cells trigger the dissociation of the endothelial receptor phosphatase VE-PTP from VE-cadherin. Blood. 2013;122(14):2512-2522.
VE-PTP maintains the endothelial barrier via plakoglobin and becomes dissociated from VE-cadherin by leukocytes and by VEGF
Nottebaum AF, Cagna G, Winderlich M, et al. VE-PTP maintains the endothelial barrier via plakoglobin and becomes dissociated from VE-cadherin by leukocytes and by VEGF. J Exp Med. 2008;205(12):2929-2945.
Increase in vascular permeability induced by leukotriene B4 and the role of polymorphonuclear leukocytes
Björk J, Hedqvist P, Arfors K-E. Increase in vascular permeability induced by leukotriene B4 and the role of polymorphonuclear leukocytes. Inflammation. 1982;6(2):189-200.
Stabilizing the VE-cadherin-catenin complex blocks leukocyte extravasation and vascular permeability
Schulte D, Kuppers V, Dartsch N, et al. Stabilizing the VE-cadherin-catenin complex blocks leukocyte extravasation and vascular permeability. EMBO J. 2011;19 (30):4157-4170.
Dissociation of VE-PTP from VE-cadherin is required for leukocyte extravasation and for VEGF-induced vascular permeability in vivo
Broermann A, Winderlich M, Block H, et al. Dissociation of VE-PTP from VE-cadherin is required for leukocyte extravasation and for VEGF-induced vascular permeability in vivo. J Exp Med. 2011;208(12):2393-2401.