Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.

Journal of the American Heart Association

Volumn 2, Issue 4, 2013, Pages

  Wang, Wang ^a   McKinnie, Shaun M K ^b   Patel, Vaibhav B ^b   Haddad, George ^b   Wang, Zuocheng ^b   Zhabyeyev, Pavel ^b   Das, Subhash K ^b   Basu, Ratnadeep ^b   McLean, Brent ^b   Kandalam, Vijay ^b   Penninger, Josef M ^b   Kassiri, Zamaneh ^b   Vederas, John C ^b   Murray, Allan G ^b   Oudit, Gavin Y ^b  

^a UNIVERSITY OF ALBERTA   (Canada)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

APLN PROTEIN, HUMAN; APLN PROTEIN, MOUSE; CARDIOVASCULAR AGENT; PEPTIDE; SIGNAL PEPTIDE;

ANGIOGENESIS; ANIMAL; C57BL MOUSE; CONVALESCENCE; DEFICIENCY; DISEASE MODEL; DRUG EFFECTS; ENDOTHELIUM CELL; GENETICS; HEART FAILURE; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE; HEART VENTRICLE REMODELING; HUMAN; KNOCKOUT MOUSE; MALE; METABOLISM; MORTALITY; MOUSE; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; PATHOLOGY; PATHOPHYSIOLOGY; PREVENTION AND CONTROL; STEM CELL; TIME;

ANIMALS; CARDIOVASCULAR AGENTS; DISEASE MODELS, ANIMAL; ENDOTHELIAL CELLS; HEART FAILURE; HUMANS; INTERCELLULAR SIGNALING PEPTIDES AND PROTEINS; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; NEOVASCULARIZATION, PHYSIOLOGIC; PEPTIDES; RECOVERY OF FUNCTION; STEM CELLS; TIME FACTORS; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR FUNCTION, LEFT; VENTRICULAR REMODELING;

EID: 84887527968     PISSN: None     EISSN: 20479980     Source Type: Journal    
DOI: 10.1161/JAHA.113.000249     Document Type: Article

References (0)