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Volumn 2, Issue 4, 2013, Pages
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Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.
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Author keywords
[No Author keywords available]
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Indexed keywords
APLN PROTEIN, HUMAN;
APLN PROTEIN, MOUSE;
CARDIOVASCULAR AGENT;
PEPTIDE;
SIGNAL PEPTIDE;
ANGIOGENESIS;
ANIMAL;
C57BL MOUSE;
CONVALESCENCE;
DEFICIENCY;
DISEASE MODEL;
DRUG EFFECTS;
ENDOTHELIUM CELL;
GENETICS;
HEART FAILURE;
HEART LEFT VENTRICLE FUNCTION;
HEART MUSCLE;
HEART VENTRICLE REMODELING;
HUMAN;
KNOCKOUT MOUSE;
MALE;
METABOLISM;
MORTALITY;
MOUSE;
MYOCARDIAL ISCHEMIA;
MYOCARDIAL REPERFUSION INJURY;
PATHOLOGY;
PATHOPHYSIOLOGY;
PREVENTION AND CONTROL;
STEM CELL;
TIME;
ANIMALS;
CARDIOVASCULAR AGENTS;
DISEASE MODELS, ANIMAL;
ENDOTHELIAL CELLS;
HEART FAILURE;
HUMANS;
INTERCELLULAR SIGNALING PEPTIDES AND PROTEINS;
MALE;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
MYOCARDIAL ISCHEMIA;
MYOCARDIAL REPERFUSION INJURY;
MYOCARDIUM;
NEOVASCULARIZATION, PHYSIOLOGIC;
PEPTIDES;
RECOVERY OF FUNCTION;
STEM CELLS;
TIME FACTORS;
VENTRICULAR DYSFUNCTION, LEFT;
VENTRICULAR FUNCTION, LEFT;
VENTRICULAR REMODELING;
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EID: 84887527968
PISSN: None
EISSN: 20479980
Source Type: Journal
DOI: 10.1161/JAHA.113.000249 Document Type: Article |
Times cited : (175)
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References (0)
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