Diabetes: Macrophages mediate β-cell loss in T2DM

Nature Reviews Endocrinology

Volumn 9, Issue 11, 2013, Pages 626-

  Kugelberg, Elisabeth ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

CANNABINOID 1 RECEPTOR; CRYOPYRIN; ENDOCANNABINOID; INFLAMMASOME;

APOPTOSIS; CELL DEATH; CELL ISOLATION; CELL LOSS; HUMAN; MACROPHAGE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; NOTE; PANCREAS ISLET BETA CELL; PANCREAS ISLET CELL FUNCTION; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; ARACHIDONIC ACIDS; CARRIER PROTEINS; DIABETES MELLITUS, TYPE 2; ENDOCANNABINOIDS; HUMANS; INFLAMMASOMES; INSULIN-SECRETING CELLS; MACROPHAGES; MALE; POLYUNSATURATED ALKAMIDES;

EID: 84886597384     PISSN: 17595029     EISSN: 17595037     Source Type: Journal    
DOI: 10.1038/nrendo.2013.177     Document Type: Note

References (1)

1. Jourdan, T. et al. Activation of the Nlrp3 inflammasome in infiltrating macrophages by endocannabinoids mediates β-cell loss in type 2 diabetes. Nat. Med. doi:10.1038/nm.3265