TRP53 activates a global autophagy program to promote tumor suppression

Autophagy

Volumn 9, Issue 9, 2013, Pages 1440-1442

  Broz, Daniela Kenzelmann ^a,b   Attardi, Laura D ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF BASEL   (Switzerland)

Author keywords

Apoptosis; Autophagy; ChIP seq; P53; RNA seq; Tumor suppression

Indexed keywords

TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR TRP53; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; AUTOPHAGY; CANCER INHIBITION; CANCER SURVIVAL; CELL CYCLE ARREST; DNA DAMAGE; GENETIC ASSOCIATION; NONHUMAN; TRANSCRIPTION INITIATION;

APOPTOSIS; AUTOPHAGY; CHIP-SEQ; P53; RNA-SEQ; TUMOR SUPPRESSION;

ANIMALS; APOPTOSIS; AUTOPHAGY; CELL CYCLE; DNA DAMAGE; DNA-BINDING PROTEINS; HUMANS; MICE; MODELS, BIOLOGICAL; NEOPLASMS; NUCLEAR PROTEINS; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 84884331483     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.25833     Document Type: Article

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